61. |
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62. |
- Angere, Staffan, et al.
(författare)
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Inquiry and deliberation in judicial systems : The problem of jury size
- 2016
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Ingår i: Perspectives on Interrogative Models of Inquiry : Developments in Inquiry and Questions - Developments in Inquiry and Questions. - Cham : Springer International Publishing. - 9783319207629 - 9783319207612 ; , s. 35-56
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Bokkapitel (refereegranskat)abstract
- We raise the question whether there is a rigorous argument favoring one jury system over another. We provide a Bayesian model of deliberating juries that allows for computer simulation for the purpose of studying the effect of jury size and required majority on the quality of jury decision making. We introduce the idea of jury value (J-value), a kind of epistemic value which takes into account the unique characteristics and asymmetries involved in jury voting. Our computer simulations indicate that requiring more than a > 50 % majority should be avoided. Moreover, while it is in principle always better to have a larger jury, given a > 50 % required majority, the value of having more than 12–15 jurors is likely to be negligible. Finally, we provide a formula for calculating the optimal jury size given the cost, economic or otherwise, of adding another juror.
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63. |
- Angere, Staffan, et al.
(författare)
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Publish Late, Publish Rarely! : Network Density and Group Performance in Scientific Communication
- 2017
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Ingår i: Scientific Collaboration and Collective Knowledge. - : Oxford University Press. - 9780190680534 ; , s. 34-62
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Bokkapitel (refereegranskat)abstract
- Research programs regularly compete to achieve the same goal, such as the discovery of the structure of DNA or the construction of a TEA laser. The more the competing programs share information, the faster the goal is likely to be reached, to society’s benefit. But the “priority rule”-the scientific norm according to which the first program to reach the goal in question must receive all the credit for the achievement-provides a powerful disincentive for programs to share information. How, then, is the clash between social and individual interest resolved in scientific practice? This chapter investigates what Robert Merton called science’s “communist” norm, which mandates universal sharing of knowledge, and uses mathematical models of discovery to argue that a communist regime may be on the whole advantageous and fair to all parties, and so might be implemented by a social contract that all scientists would be willing to sign.
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64. |
- Anrup, Roland, et al.
(författare)
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Centrala universitetsvärden hotas av bolagiseringsidén
- 2013
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Ingår i: Dagens nyheter. - 1101-2447.
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Tidskriftsartikel (populärvet., debatt m.m.)abstract
- Högskolestiftelser. Förslaget att driva svenska universitet i stiftelseform öppnar för bolagisering. Men det är ingen riktig utredning, utan en politisk pamflett utan eftertanke. Privatisering av universitet hotar både oberoendet, forskningskvaliteten och samhällsnyttan, skriver 36 forskare vid svenska högskolor och universitet.
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65. |
- Armbruster, Henriet, et al.
(författare)
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On-board Conversion of Alcohol to Ethers for diesel Engines
- 2000
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Ingår i: ISAF XIII Paper. International Symposium on Alcohol Fuels. Implementing the Transition to a Sustainable Transport System, Stockholm 3-6 July 2000..
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Konferensbidrag (refereegranskat)
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66. |
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67. |
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68. |
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69. |
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70. |
- Bereketoglu, Ceyhun, et al.
(författare)
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The brominated flame retardants TBECH and DPTE alter prostate growth, histology and gene expression patterns in the mouse
- 2021
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Ingår i: Reproductive Toxicology. - : Elsevier. - 0890-6238 .- 1873-1708. ; 102, s. 43-55
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Tidskriftsartikel (refereegranskat)abstract
- The brominated flame retardants (BFRs), 1,2-dibromo-4-(1,2 dibromoethyl)cyclohexane (TBECH) and 2,3-dibromopropyl-2,4,6-tribromophenyl ether (DPTE) bind to the androgen receptor (AR). In vitro bioassays have shown that TBECH is a potent androgen agonist while DPTE is a potent AR antagonist. Both TBECH and DPTE alter gene expression associated with AR regulation. However, it remains to be determined if TBECH and DPTE can affect the prostate. For this reason, we exposed CD1 mice to a 1:1 mixture of TBECH diastereomers α and β, a 1:1 mixture of γ and δ, and to DPTE, and tested their effects on prostate growth, histology and gene expression profiles. Castrated (C) mice were used to study the androgenic effects of TBECHαβ and TBECHγδ while the antagonistic effects of DPTE were studied in non-castrated (NC) mice. We observed that testosterone and TBECHγδ increased body and prostate weights while TBECHαβ affected neither of them; and that DPTE had no effect on body weight but reduced prostate weight drastically. Histomorphometric analysis of the prostate revealed epithelial and glandular alterations in the TBECHγδ group comparable to those in testosterone group while alterations in the TBECHαβ group were less pronounced. DPTE displayed androgen antagonist activity reminiscent of castration. The transcription profile of the prostate was altered by castration and exposure to testosterone and to TBECHγδ reversed several of these changes. Testosterone and TBECHγδ also regulated the expression of several androgen responsive genes implicated in prostate growth and cancer. While DPTE resulted in a drastic reduction in prostate weight, it only affected a small number of genes. The results indicate that TBECHγδ and DPTE are of high human health concern as they may contribute to changes in prostate growth, histology and function.
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