61. |
- Sjogren, B, et al.
(författare)
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Ischemic heart disease in female cleaners
- 2003
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Ingår i: International journal of occupational and environmental health. - : Informa UK Limited. - 1077-3525 .- 2049-3967. ; 9:2, s. 134-137
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Tidskriftsartikel (refereegranskat)
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62. |
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63. |
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64. |
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65. |
- Sjogren, B, et al.
(författare)
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Welding and ischemic heart disease
- 2002
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Ingår i: International journal of occupational and environmental health. - 1077-3525. ; 8:4, s. 309-311
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Tidskriftsartikel (refereegranskat)
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66. |
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67. |
- Sjogren, C, et al.
(författare)
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Introduction. DNA damage and repair
- 2014
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Ingår i: Experimental cell research. - : Elsevier BV. - 1090-2422 .- 0014-4827. ; 329:1, s. 1-1
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Tidskriftsartikel (övrigt vetenskapligt/konstnärligt)
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68. |
- Sjogren, H., et al.
(författare)
-
Cytogenetic and spectral karyotype analyses of benign and malignant cartilage tumours
- 2004
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Ingår i: International Journal of Oncology. - 1019-6439. ; 24:6, s. 1385-91
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Tidskriftsartikel (refereegranskat)abstract
- To date, there have been few studies published on benign and malignant cartilage tumours using high resolution molecular cytogenetic techniques such as spectral karyotyping (SKY). In this study we have used a combination of chromosome banding, SKY and FISH to characterize the chromosomal pattern in 18 benign and malignant cartilage tumours and one small cell osteosarcoma with mesenchymal chondrosarcoma-like features. Clonal structural and/or numerical aberrations were detected in 14 of these tumours. All chondroblastomas and the chondromyxoid fibroma had diploid or near-diploid karyotypes with often relatively simple karyotypes. Although no consistent abnormalities were detected in the chondroblastomas, recurrent breakpoints were found at 2q35, 3q21-23, and 18q21. The chondromyxoid fibroma had an inv(6)(p25q13) as the sole anomaly, suggesting that this is a primary abnormality characteristic of this entity. The karyotypic findings in the chondrosarcomas were, as a rule, more complex than those in the benign tumours. A typical feature was the frequent occurrence of unbalanced rearrangements leading to genomic imbalances with losses and gains of certain chromosomes or chromosome regions. The following breakpoints were recurrent: Xq21, 6p10, 9p13, 20p11 and 22q11-12. Despite the use of high-resolution molecular cytogenetic techniques, we were not able to identify any consistent abnormalities in chondrosarcomas, suggesting that tumour-specific chromosome changes are not likely to be found in this group of tumours.
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69. |
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70. |
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