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Träfflista för sökning "WFRF:(Rasmussen A) srt2:(1995-1999)"

Sökning: WFRF:(Rasmussen A) > (1995-1999)

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  • Asztalos, S J, et al. (författare)
  • Spin yields of neutron-rich nuclei from deep inelastic reactions
  • 1999
  • Ingår i: Physical Review C. Nuclear Physics. - 0556-2813 .- 1089-490X. ; 60:4
  • Tidskriftsartikel (refereegranskat)abstract
    • The potential for using deep inelastic reactions to populate high-spin states in neutron-rich nuclei is studied in a series of experiments using GAMMASPHERE for gamma-ray detection and a silicon strip detector for measuring the angles of projectilelike and targetlike fragments. In three experiments 61 new transitions up to a maximum spin of 22 (h) over bar in 12 neutron-rich rare-earth nuclei were found. We observe that gamma-ray yields as a function of spin are flatter for all neutron transfer products than for inelastic excitation of either the projectile or target nucleus. Calculations are presented which indicate that this difference cannot be accounted for by quasielastic processes, but more likely are the result of larger energy loss processes, such as deep inelastic reactions. [S0556-2813(99)06009-4].
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  • Hillerdal, Gunnar, et al. (författare)
  • Pleural disease during treatment with bromocriptine in patients previously exposed to asbestos
  • 1997
  • Ingår i: European Respiratory Journal. - : European Respiratory Society (ERS). - 0903-1936 .- 1399-3003. ; 10:12, s. 2711-2715
  • Tidskriftsartikel (refereegranskat)abstract
    • Bromocriptine, which is used in the treatment of Parkinson's disease, can cause adverse pleuropulmonary reactions. Exposure to asbestos can result in similar lesions. Fifteen patients with former exposure to asbestos, who developed pleural fibrosis after treatment with bromocriptine, were observed independently in Sweden (11 patients) and Australia (four patients). The patients complained of malaise, often associated with weight loss, dyspnoea, and a disturbing cough. Laboratory values included increased erythrocyte sedimentation rate and a low haemoglobin level. Lung function tests showed a restrictive lung function defect. Chest radiographs showed bilateral pleural fibrosis, with small amounts of fluid in some cases. Soon after bromocriptine was withdrawn, the patients improved clinically, and the laboratory values returned to normal. However, in most cases, pleural fibrosis and a restrictive lung function defect persisted to some extent. In conclusion, in patients who develop pleuropulmonary fibrosis whilst being treated with bromocriptine, former exposure to asbestos should be investigated. Conversely, when pleural changes develop in a patient on bromocriptine and with prior exposure to asbestos, the possible causative role of the drug should be discussed. Special follow-up may be indicated when bromocriptine is planned in a patient with previous asbestos exposure, and if symptoms or signs of pleural fibrosis develop, bromocriptine withdrawal should be considered.
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