Neuropeptides in hyperthermia

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Fältnamn	Indikatorer	Metadata
000		03211naa a2200409 4500
001		oai:DiVA.org:uu-102864
003		SwePub
008		090512s2007 \| \|\|\|\|\|\|\|\|\|\|\|000 \|\|eng\|
024	7	a https://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-1028642 URI
024	7	a https://doi.org/10.1016/S0079-6123(06)62014-12 DOI
040		a (SwePub)uu
041		a engb eng
042		9 SwePub
072	7	a ref2 swepub-contenttype
072	7	a for2 swepub-publicationtype
100	1	a Nyberg, Fredu Uppsala universitet,Avdelningen för biologisk beroendeforskning4 aut0 (Swepub:uu)frednybe
245	1 0	a Neuropeptides in hyperthermia
264	1	c 2007
338		a print2 rdacarrier
520		a Brain damage as a result of hyperthermia or heat-stress has been the focus of attention in many areas of neuroscience in recent years. Heat-induced alterations in structural components of the central nervous system (CNS) will obviously also influence the relevant transmitter systems, which may be involved in a variety of different behaviors. Indeed, many studies have indicated that excitatory amino acids, and monoaminergic and peptidergic systems are affected during hyperthermia. This chapter will address past and current research on various neuropeptides that have been implicated in the consequences of hyperthermia and various other heat disorders. However, considering the large and even increasing number of identified neuroactive peptides, it is necessary to limit this chapter to a few peptides or peptide systems, which have received particular attention in relation to hyperthermia. Among these are the opioid peptides, the tachykinins, calcitonin gene-related peptide (CGRP), and peptides belonging to the angiotensin system. Most of these neuropeptides are not only affected by hyperthermia and abnormal alterations in the body temperature but also are involved in the endogenous mechanisms of regulating body temperature. This review does not endeavor to fully cover the field but it does aim to give the reader an idea of how various neuropeptides may be involved in the control of body heat and how peptidergic systems are affected during various thermal changes, including both immediate and long-term consequences.
650	7	a MEDICIN OCH HÄLSOVETENSKAPx Medicinska och farmaceutiska grundvetenskaperx Farmaceutiska vetenskaper0 (SwePub)301012 hsv//swe
650	7	a MEDICAL AND HEALTH SCIENCESx Basic Medicinex Pharmaceutical Sciences0 (SwePub)301012 hsv//eng
653		a angiotensin
653		a calcitonin gene-related peptides (CGRP)
653		a dynorphin
653		a heat stress
653		a hyperthermia
653		a neuropeptides
653		a opiod peptides
653		a substance P
653		a PHARMACY
653		a FARMACI
700	1	a Hallberg, Mathiasu Uppsala universitet,Avdelningen för biologisk beroendeforskning4 aut0 (Swepub:uu)mha03066
710	2	a Uppsala universitetb Avdelningen för biologisk beroendeforskning4 org
773	0	t Progress in Brain Researchg 162, s. 277-293q 162<277-293x 0079-6123x 1875-7855
856	4 8	u https://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-102864
856	4 8	u https://doi.org/10.1016/S0079-6123(06)62014-1

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