Sökning: onr:"swepub:oai:prod.swepub.kib.ki.se:150544241" > The whole blood tra...
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000 | 16308naa a2205545 4500 | |
001 | oai:prod.swepub.kib.ki.se:150544241 | |
003 | SwePub | |
008 | 240913s2022 | |||||||||||000 ||eng| | |
024 | 7 | a http://kipublications.ki.se/Default.aspx?queryparsed=id:1505442412 URI |
024 | 7 | a https://doi.org/10.1038/s41467-022-32276-22 DOI |
040 | a (SwePub)ki | |
041 | a engb eng | |
042 | 9 SwePub | |
072 | 7 | a ref2 swepub-contenttype |
072 | 7 | a art2 swepub-publicationtype |
100 | 1 | a Wang, QBS4 aut |
245 | 1 0 | a The whole blood transcriptional regulation landscape in 465 COVID-19 infected samples from Japan COVID-19 Task Force |
264 | c 2022-08-22 | |
264 | 1 | b Springer Science and Business Media LLC,c 2022 |
520 | a Coronavirus disease 2019 (COVID-19) is a recently-emerged infectious disease that has caused millions of deaths, where comprehensive understanding of disease mechanisms is still unestablished. In particular, studies of gene expression dynamics and regulation landscape in COVID-19 infected individuals are limited. Here, we report on a thorough analysis of whole blood RNA-seq data from 465 genotyped samples from the Japan COVID-19 Task Force, including 359 severe and 106 non-severe COVID-19 cases. We discover 1169 putative causal expression quantitative trait loci (eQTLs) including 34 possible colocalizations with biobank fine-mapping results of hematopoietic traits in a Japanese population, 1549 putative causal splice QTLs (sQTLs; e.g. two independent sQTLs at TOR1AIP1), as well as biologically interpretable trans-eQTL examples (e.g., REST and STING1), all fine-mapped at single variant resolution. We perform differential gene expression analysis to elucidate 198 genes with increased expression in severe COVID-19 cases and enriched for innate immune-related functions. Finally, we evaluate the limited but non-zero effect of COVID-19 phenotype on eQTL discovery, and highlight the presence of COVID-19 severity-interaction eQTLs (ieQTLs; e.g., CLEC4C and MYBL2). Our study provides a comprehensive catalog of whole blood regulatory variants in Japanese, as well as a reference for transcriptional landscapes in response to COVID-19 infection. | |
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700 | 1 | a Takahashi, H4 aut |
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700 | 1 | a Saito, Y4 aut |
700 | 1 | a Seike, M4 aut |
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700 | 1 | a Nakano, C4 aut |
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700 | 1 | a Hirouchi, T4 aut |
700 | 1 | a Makino, S4 aut |
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700 | 1 | a Nannya, Y4 aut |
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700 | 1 | a Takano, T4 aut |
700 | 1 | a Katayama, K4 aut |
700 | 1 | a Ai, M4 aut |
700 | 1 | a Kumanogoh, A4 aut |
700 | 1 | a Sato, T4 aut |
700 | 1 | a Hasegawa, N4 aut |
700 | 1 | a Tokunaga, K4 aut |
700 | 1 | a Ishii, M4 aut |
700 | 1 | a Koike, R4 aut |
700 | 1 | a Kitagawa, Y4 aut |
700 | 1 | a Kimura, A4 aut |
700 | 1 | a Imoto, S4 aut |
700 | 1 | a Miyano, S4 aut |
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773 | 0 | t Nature communicationsd : Springer Science and Business Media LLCg 13:1, s. 4830-q 13:1<4830-x 2041-1723 |
856 | 4 8 | u http://kipublications.ki.se/Default.aspx?queryparsed=id:150544241 |
856 | 4 8 | u https://doi.org/10.1038/s41467-022-32276-2 |
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