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Träfflista för sökning "L773:1551 4005 OR L773:1538 4101 "

Search: L773:1551 4005 OR L773:1538 4101

  • Result 1-10 of 158
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  • Agarwal, Prasoon, et al. (author)
  • Growth signals employ CGGBP1 to suppress transcription of Alu-SINEs
  • 2016
  • In: Cell Cycle. - : Informa UK Limited. - 1538-4101 .- 1551-4005. ; 15:12, s. 1558-1571
  • Journal article (peer-reviewed)abstract
    • CGGBP1 (CGG triplet repeat-binding protein 1) regulates cell proliferation, stress response,cytokinesis, telomeric integrity and transcription. It could affect these processes by modulatingtarget gene expression under different conditions. Identification of CGGBP1-target genes andtheir regulation could reveal how a transcription regulator affects such diverse cellular processes.Here we describe the mechanisms of differential gene expression regulation by CGGBP1 inquiescent or growing cells. By studying global gene expression patterns and genome-wide DNAbindingpatterns of CGGBP1, we show that a possible mechanism through which it affects theexpression of RNA Pol II-transcribed genes in trans depends on Alu RNA. We also show that itregulates Alu transcription in cis by binding to Alu promoter. Our results also indicate thatpotential phosphorylation of CGGBP1 upon growth stimulation facilitates its nuclear retention,Alu-binding and dislodging of RNA Pol III therefrom. These findings provide insights into howAlu transcription is regulated in response to growth signals.
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  • Akanda, Nesar, et al. (author)
  • Voltage-dependent anion channels (VDAC) in the plasma membrane play a critical role in apoptosis in differentiated hippocampal neurons but not in neural stem cells
  • 2008
  • In: Cell Cycle. - : Informa UK Limited. - 1538-4101 .- 1551-4005. ; 7:20, s. 3225-3234
  • Journal article (peer-reviewed)abstract
    • microRNAs (miRNAs) are small non-coding RNAs that regulate a large variety of cellular processes including differentiation, apoptosis and proliferation. Several miRNAs display defective expression patterns in human tumors with the consequent alteration of target oncogene or tumor suppressor genes. Many of these miRNAs modulate the major proliferation pathways through direct interaction with critical regulators such as RAS, PI3K/PTEN or ABL, as well as members of the retinoblastoma pathway, Cyclin-CDK complexes or cell cycle inhibitors of the INK4 or Cip/Kip families. A complex interplay between miRNAs and MYC or E2F family members also exists to modulate cell cycle-dependent transcription during normal or tumoral proliferation. The ability of miRNAs to modulate these proliferation pathways may have relevant implications not only in physiological or developmental processes but also in tumor progression or cancer therapy.
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  • Andang, M, et al. (author)
  • To go or not to go?
  • 2015
  • In: Cell cycle (Georgetown, Tex.). - : Informa UK Limited. - 1551-4005 .- 1538-4101. ; 14:8, s. 1136-1137
  • Journal article (peer-reviewed)
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  • Anderberg, C, et al. (author)
  • On the origin of cancer-associated fibroblasts
  • 2009
  • In: Cell cycle (Georgetown, Tex.). - : Informa UK Limited. - 1551-4005 .- 1538-4101. ; 8:10, s. 1461-1462
  • Journal article (other academic/artistic)
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  • Aufschnaiter, Andreas, Dr. rer. nat. 1988-, et al. (author)
  • Peroxisomal fission controls yeast life span
  • 2015
  • In: Cell Cycle. - : Taylor & Francis. - 1538-4101 .- 1551-4005. ; 14:15, s. 2389-2390
  • Journal article (peer-reviewed)
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