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1.	Jakobsson, J., et al. (author) Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1 2021 In: Autophagy. - : Informa UK Limited. - 1554-8635 .- 1554-8627. ; 17:1, s. 1-382 Journal article (peer-reviewed)
2.	Aad, G., et al. (author) 2011 swepub:Mat__t (peer-reviewed)
3.	Goncalves, Isabel, et al. (author) Evidence Supporting a Key Role of Lp-PLA2-Generated Lysophosphatidylcholine in Human Atherosclerotic Plaque Inflammation. 2012 In: Arteriosclerosis, Thrombosis and Vascular Biology. - 1524-4636. ; 32:6, s. 1505-1505 Journal article (peer-reviewed)abstract OBJECTIVE: To determine whether the level of lysophosphatidylcholine (lysoPC) generated by lipoprotein-associated phospholipase A2 (Lp-PLA2) is associated with severity of inflammation in human atherosclerotic plaques. Elevated plasma Lp-PLA2 is associated with increased cardiovascular risk. Lp-PLA2 inhibition reduces atherosclerosis. Lp-PLA2 hydrolyzes low-density lipoprotein-oxidized phospholipids generating lysoPCs. According to in vitro studies, lysoPCs are proinflammatory but the association between their generation and plaque inflammation remains unknown. METHODS AND RESULTS: Inflammatory activity in carotid plaques (162 patients) was determined immunohistochemically and by analyzing cytokines in homogenates (multiplex immunoassay). LysoPCs were quantified using mass spectrometry and Lp-PLA2 and the lysoPC metabolite lysophosphatidic acid (LPA) by ELISA. There was a strong correlation among lysoPC 16:0, 18:0, 18:1, LPA, and Lp-PLA2 in plaques. LysoPC 16:0, 18:0, 18:1, LPA, and Lp-PLA2 correlated with interleukin-1β, interleukin-6, monocyte chemoattractant protein-1, macrophage inflammatory protein-1β, regulated on activation normal T-cell expressed and secreted, and tumor necrosis factor-α in plaques. High lysoPC and Lp-PLA2 correlated with increased plaque macrophages and lipids and with low content of smooth muscle cells, whereas LPA only correlated with plaque macrophages. Lp-PLA2, lysoPC 16:0, 18:0, and 18:1, but not LPA were higher in symptomatic than in asymptomatic plaques. CONCLUSIONS: The associations among Lp-PLA2, lysoPCs, LPA, and proinflammatory cytokines in human plaques suggest that lysoPCs play a key role in plaque inflammation and vulnerability. Our findings support Lp-PLA2 inhibition as a possible strategy for the prevention of cardiovascular disease.
4.	Aad, G., et al. (author) 2011 swepub:Mat__t (peer-reviewed)
5.	Aad, G., et al. (author) 2010 swepub:Mat__t
6.	Aad, G., et al. (author) 2012 swepub:Mat__t (peer-reviewed)
7.	Aad, G., et al. (author) A search for prompt lepton-jets in pp collisions at root s=7 TeV with the ATLAS detector 2013 Journal article (peer-reviewed)
8.	Aad, G., et al. (author) A search for t (t)over-bar resonances with the ATLAS detector in 2.05 fb(-1) of proton-proton collisions at root s=7 TeV 2012 In: European Physical Journal C. Particles and Fields. - : Springer Science and Business Media LLC. - 1434-6044 .- 1434-6052. ; 72:7 Journal article (peer-reviewed)
9.	Aad, G., et al. (author) ATLAS measurements of the properties of jets for boosted particle searches 2012 In: Physical Review D (Particles, Fields, Gravitation and Cosmology). - 1550-2368 .- 1550-7998. ; 86:7 Journal article (peer-reviewed)
10.	Aad, G., et al. (author) Combined search for the Standard Model Higgs boson in pp collisions at root s=7 TeV with the ATLAS detector 2012 In: Physical Review D (Particles, Fields, Gravitation and Cosmology). - 1550-2368 .- 1550-7998. ; 86:3 Journal article (peer-reviewed)

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Type of publication: journal article (169); doctoral thesis (1); research review (1); book chapter (1)

Type of content: peer-reviewed (288); other academic/artistic (3); pop. science, debate, etc. (1)

Author/Editor: Chen, X. (280); Adelman, J. (278); Adye, T. (278); Aleksa, M. (278); Amelung, C. (278); Arai, Y. (278); show more...; Arnaez, O. (278); Asquith, L. (278); Bachas, K. (278); Backes, M. (278); Baker, O. K. (278); Banas, E. (278); Barklow, T. (278); Beau, T. (278); Beck, H. P. (278); Bella, G. (278); Benary, O. (278); Benekos, N. (278); Berry, T. (278); Bilokon, H. (278); Black, K. M. (278); Boehler, M. (278); Bold, T. (278); Borisov, A. (278); Bulekov, O. (278); Burke, S. (278); Busato, E. (278); Cameron, D. (278); Campana, S. (278); Carli, T. (278); Caron, S. (278); Casadei, D. (278); Chen, H. (278); Cheu, E. (278); Cinca, D. (278); Cindro, V. (278); Clark, A. (278); Cote, D. (278); Cox, B. E. (278); Cranmer, K. (278); D'Auria, S. (278); Da Via, C. (278); Dai, T. (278); Dam, M. (278); Dao, V. (278); Davidek, T. (278); Dawson, I. (278); Deluca, C. (278); Dobos, D. (278); Dopke, J. (278); show less...

University: Lund University (142); Uppsala University (137); Stockholm University (136); Royal Institute of Technology (134); Karolinska Institutet (9); University of Gothenburg (5); show more...; Umeå University (5); Linköping University (4); Swedish University of Agricultural Sciences (2); University West (1); show less...

Language: English (300)

Research subject (UKÄ/SCB): Natural sciences (138); Medical and Health Sciences (11)

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