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- Shergur, J., et al.
(author)
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beta-decay studies of Sn135-137 using selective resonance laser ionization techniques
- 2002
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In: Physical Review C - Nuclear Physics. - 2469-9985 .- 2469-9993. ; 65:3
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Journal article (peer-reviewed)abstract
- The decays of the very neutron rich Sn isotopes Sn135-137 were studied at CERN/ISOLDE using isotopic and isobaric selectivity achieved by the use of a resonance ionization laser ion source and mass spectroscopy, respectively. Neutron decay rates, gamma-ray singles, and gamma-gamma coincidence data were collected as a function of time. The half-life (T-1/2) and delayed neutron emission probability (P-n) values of 135 Sn were measured to be 530(20) ms and 21(3)%, respectively. For Sn-136, a T-1/2 of 250(30) ms was determined along with a P-n value of 30(5)%. For Sn-137, a T-1/2 of 190(60) ms and a P-n value of 58(15)% were deduced. Identification of low-energy transitions in Sb-135 was made possible by comparison of laser-on and laser-off gamma-ray spectra. Those data combined with gamma-gamma coincidence spectra were used to construct a level scheme for Sb-135 that includes an unexpectedly low first excited state at 282 keV. A ground state beta branch of 33.2% was measured by following the growth and decay of the Sb-135 daughter. Shell-model calculations are consistent with the observed Sb-135 level structure and can account for a lowered first excited state.
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- Shergur J, Brown BA, Fedoseyev V, Koster U, Kratz KL, Sweryniak D, Walters WB, Wohr A, Fedorov D, Hannawald M, Hjorth-Jensen M, Mishin V, Pfeiffer B, Ressler JJ, Fynbo HOU, Hoff P, Mach H, Nilsson T, Wilhelmsen-Rolander K, Simon H, Bickley A and the ISOLD
(author)
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Beta-Decay Studies of 135-137Sn using Selective Resonance Laser Ionization Techniques
- 2002
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In: Phys. Rev.. ; C65, s. 34313-
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Journal article (peer-reviewed)
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- Shergur, J., et al.
(author)
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Decay of Sn-135,Sn-136 isolated by use of a laser ion source and evidence for a more harmonic-oscillator-like nuclear potential
- 2001
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In: Nuclear Physics A. - 0375-9474. ; 682, s. 493C-497C
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Conference paper (peer-reviewed)abstract
- The use of a resonance ionization laser ion source at CERN/ISOLDE has made it possible to study the decay of very neutron-rich Sn135-137. The decay of Sn-135 is found to populate low-energy levels in Sb-135 via direct beta decay and the first excited state in Sb-134 by beta-delayed neutron emission. The level structure of Sb-135 Will be discussed and a possible signature for a more diffuse nuclear surface considered.
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- Andersson, Jenny, et al.
(author)
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Absence of the Lhcb1 and Lhcb2 proteins of the light-harvesting complex of photosystem II - effects on photosynthesis, grana stacking and fitness
- 2003
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In: The Plant Journal. - : Wiley. - 0960-7412 .- 1365-313X. ; 35:3, s. 350-361
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Journal article (peer-reviewed)abstract
- We have constructed Arabidopsis thaliana plants that are virtually devoid of the major light-harvesting complex, LHC II. This was accomplished by introducing the Lhcb2.1 coding region in the antisense orientation into the genome by Agrobacterium-mediated transformation. Lhcb1 and Lhcb2 were absent, while Lhcb3, a protein present in LHC II associated with photosystem (PS) II, was retained. Plants had a pale green appearance and showed reduced chlorophyll content and an elevated chlorophyll a/b ratio. The content of PS II reaction centres was unchanged on a leaf area basis, but there was evidence for increases in the relative levels of other light harvesting proteins, notably CP26, associated with PS II, and Lhca4, associated with PS I. Electron microscopy showed the presence of grana. Photosynthetic rates at saturating irradiance were the same in wild-type and antisense plants, but there was a 10-15% reduction in quantum yield that reflected the decrease in light absorption by the leaf. The antisense plants were not able to perform state transitions, and their capacity for non-photochemical quenching was reduced. There was no difference in growth between wild-type and antisense plants under controlled climate conditions, but the antisense plants performed worse compared to the wild type in the field, with decreases in seed production of up to 70%.
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- Andreyev, A. N., et al.
(author)
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Shape-changing particle decays of Bi-185 and structure of the lightest odd-mass Bi isotopes
- 2004
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In: Physical Review C. Nuclear Physics. - 0556-2813 .- 1089-490X. ; 69:5, s. 054308-
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Journal article (peer-reviewed)abstract
- Proton and alpha decay of the proton-rich nuclide Bi-185 has been restudied in more detail in the complete fusion reaction Nb-93(Mo-95, 3n)Bi-185 at the velocity filter SHIP. The observed decay pattern of Bi-185 and of the heavier odd-mass isotopes Bi-187,Bi-189,Bi-191,Bi-193 are interpreted based on potential-energy surface calculations. It is shown that the experimental systematics of the particle decays and of the excited states in these nuclei (where known) can be explained by the prolate-oblate shape co-existence at low excitation energy. The observed state in Bi-185 is proposed to be of prolate nature, which is in contrast with the previously proposed oblate interpretation.
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- Popat, S, et al.
(author)
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Variation in the CTLA4/CD28 gene region confers an increased risk of coeliac disease.
- 2002
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In: Annals of human genetics. - 0003-4800 .- 1469-1809. ; 66:Pt 2, s. 125-37
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Journal article (peer-reviewed)abstract
- Susceptibility to coeliac disease involves HLA and non-HLA-linked genes. The CTLA4/CD28 gene region encodes immune regulatory T-cell surface molecules and is a strong candidate as a susceptibility locus. We evaluated CTLA4/CD28 in coeliac disease by genetic linkage and association and combined our findings with published studies through a meta-analysis. 116 multiplex families were genotyped across CTLA4/CD28 using eight markers. The contribution of CTLA4/CD28 to coeliac disease was assessed by non-parametric linkage and association analyses. Seven studies were identified that had evaluated the relationship between CTLA4/CD28 and coeliac disease and a pooled analysis of data undertaken. In our study there was evidence for a relationship between variation in the CTLA4/CD28 region and coeliac disease by linkage and association analyses. However, the findings did not attain formal statistical significance (p = 0.004 and 0.039, respectively). Pooling findings with published results showed significant evidence for linkage (504 families) and association (940 families): p values, 0.0001 and 0.0014 at D2S2214, respectively, and 0.0008 and 0.0006 at D2S116, respectively. These findings suggest that variation in the CD28/CTLA4 gene region is a determinant of coeliac disease susceptibility. Dissecting the sequence variation underlying this relationship will depend on further analyses utilising denser sets of markers.
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