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61.	Jiang, X, et al. (author) Publisher Correction: Shared heritability and functional enrichment across six solid cancers 2019 In: Nature communications. - : Springer Science and Business Media LLC. - 2041-1723. ; 10:1, s. 4386- Journal article (other academic/artistic)abstract An amendment to this paper has been published and can be accessed via a link at the top of the paper.
62.	Kar, SP, et al. (author) Genome-Wide Meta-Analyses of Breast, Ovarian, and Prostate Cancer Association Studies Identify Multiple New Susceptibility Loci Shared by at Least Two Cancer Types 2016 In: Cancer discovery. - : AMER ASSOC CANCER RESEARCH. - 2159-8290 .- 2159-8274. ; 6:9, s. 1052-1067 Journal article (peer-reviewed)
63.	Kentistou, KA, et al. (author) Understanding the genetic complexity of puberty timing across the allele frequency spectrum 2023 In: medRxiv : the preprint server for health sciences. Journal article (other academic/artistic)
64.	Kilpelainen, TO, et al. (author) Multi-ancestry study of blood lipid levels identifies four loci interacting with physical activity 2019 In: Nature communications. - London : Springer Science and Business Media LLC. - 2041-1723. ; 10:1, s. 376- Journal article (peer-reviewed)abstract Many genetic loci affect circulating lipid levels, but it remains unknown whether lifestyle factors, such as physical activity, modify these genetic effects. To identify lipid loci interacting with physical activity, we performed genome-wide analyses of circulating HDL cholesterol, LDL cholesterol, and triglyceride levels in up to 120,979 individuals of European, African, Asian, Hispanic, and Brazilian ancestry, with follow-up of suggestive associations in an additional 131,012 individuals. We find four loci, in/near CLASP1, LHX1, SNTA1, and CNTNAP2, that are associated with circulating lipid levels through interaction with physical activity; higher levels of physical activity enhance the HDL cholesterol-increasing effects of the CLASP1, LHX1, and SNTA1 loci and attenuate the LDL cholesterol-increasing effect of the CNTNAP2 locus. The CLASP1, LHX1, and SNTA1 regions harbor genes linked to muscle function and lipid metabolism. Our results elucidate the role of physical activity interactions in the genetic contribution to blood lipid levels.
65.	Kuchenbaecker, KB, et al. (author) Identification of six new susceptibility loci for invasive epithelial ovarian cancer 2015 In: Nature genetics. - : Springer Science and Business Media LLC. - 1546-1718 .- 1061-4036. ; 47:2, s. 164-171 Journal article (peer-reviewed)
66.	Lambrechts, D, et al. (author) 11q13 is a susceptibility locus for hormone receptor positive breast cancer 2012 In: Human mutation. - : Hindawi Limited. - 1098-1004 .- 1059-7794. ; 33:7, s. 1123-1132 Journal article (peer-reviewed)
67.	Li, J, et al. (author) 2q36.3 is associated with prognosis for oestrogen receptor-negative breast cancer patients treated with chemotherapy 2014 In: Nature communications. - : Springer Science and Business Media LLC. - 2041-1723. ; 5, s. 4051- Journal article (peer-reviewed)
68.	Liu, JJ, et al. (author) rs2735383, located at a microRNA binding site in the 3'UTR of NBS1, is not associated with breast cancer risk 2016 In: Scientific reports. - : Springer Science and Business Media LLC. - 2045-2322. ; 6, s. 36874- Journal article (peer-reviewed)abstract NBS1, also known as NBN, plays an important role in maintaining genomic stability. Interestingly, rs2735383 G > C, located in a microRNA binding site in the 3′-untranslated region (UTR) of NBS1, was shown to be associated with increased susceptibility to lung and colorectal cancer. However, the relation between rs2735383 and susceptibility to breast cancer is not yet clear. Therefore, we genotyped rs2735383 in 1,170 familial non-BRCA1/2 breast cancer cases and 1,077 controls using PCR-based restriction fragment length polymorphism (RFLP-PCR) analysis, but found no association between rs2735383CC and breast cancer risk (OR = 1.214, 95% CI = 0.936–1.574, P = 0.144). Because we could not exclude a small effect size due to a limited sample size, we further analyzed imputed rs2735383 genotypes (r2 > 0.999) of 47,640 breast cancer cases and 46,656 controls from the Breast Cancer Association Consortium (BCAC). However, rs2735383CC was not associated with overall breast cancer risk in European (OR = 1.014, 95% CI = 0.969–1.060, P = 0.556) nor in Asian women (OR = 0.998, 95% CI = 0.905–1.100, P = 0.961). Subgroup analyses by age, age at menarche, age at menopause, menopausal status, number of pregnancies, breast feeding, family history and receptor status also did not reveal a significant association. This study therefore does not support the involvement of the genotype at NBS1 rs2735383 in breast cancer susceptibility.
69.	Mavaddat, N, et al. (author) Polygenic Risk Scores for Prediction of Breast Cancer and Breast Cancer Subtypes 2019 In: American journal of human genetics. - : Elsevier BV. - 1537-6605 .- 0002-9297. ; 104:1, s. 21-34 Journal article (peer-reviewed)
70.	Michailidou, K, et al. (author) Association analysis identifies 65 new breast cancer risk loci 2017 In: Nature. - : Springer Science and Business Media LLC. - 1476-4687 .- 0028-0836. ; 551:7678, s. 92- Journal article (peer-reviewed)

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Result 61-70 of 172

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Type of publication: journal article (162); conference paper (1)

Type of content: peer-reviewed (136); other academic/artistic (27)

Author/Editor: Brenner, H (45); Hall, P (40); Fasching, PA (39); Chang-Claude, J (39); Zheng, W. (38); Beckmann, MW (38); show more...; Wang, Q. (37); Southey, MC (37); Couch, FJ (37); Giles, GG (36); Dunning, AM (36); Garcia-Closas, M (36); Hopper, JL (35); Czene, K (34); Milne, RL (34); Cox, A (34); Benitez, J. (32); Gudnason, V (32); Lambrechts, D (32); Anton-Culver, H (31); Hamann, U (31); Mannermaa, A (31); Jonas, JB (30); Dennis, J (30); Lindblom, A (30); Andrulis, IL (30); Shu, XO (29); Ikram, MA (29); Schmidt, MK (29); Ekici, AB (29); Burwinkel, B (29); Kosma, VM (29); Lubinski, J (29); Blomqvist, C (28); Radice, P (28); Bojesen, SE (28); Nordestgaard, BG (27); Lissowska, J (27); Jakubowska, A (27); Brauch, H (26); Peto, J (26); Haiman, CA (26); Devilee, P (26); Peterlongo, P (25); Cheng, CY (25); Wong, TY (25); Michailidou, K (25); Bolla, MK (25); Margolin, S (25); Winqvist, R (25); show less...

University: Karolinska Institutet (159); Lund University (48); Uppsala University (38); Umeå University (24); University of Gothenburg (15); Linköping University (6); show more...; Högskolan Dalarna (6); University of Skövde (5); Stockholm University (4); Swedish University of Agricultural Sciences (2); Stockholm School of Economics (1); Chalmers University of Technology (1); show less...

Language: English (172)

Research subject (UKÄ/SCB): Medical and Health Sciences (62); Natural sciences (13); Social Sciences (1)

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