1. |
- Laursen, Brett, et al.
(author)
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Friend influence over adolescent problem behaviors as a function of relative peer acceptance : to be liked is to be emulated
- 2012
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In: Journal of Abnormal Psychology. - : American Psychological Association (APA). - 0021-843X .- 1939-1846. ; 121:1, s. 88-94
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Journal article (peer-reviewed)abstract
- Friend influence over alcohol intoxication and delinquent behavior was examined as a function of relative peer acceptance in a 3-year study of Swedish youth (N = 184 girls, 145 boys). Participants were in the first year of secondary school (7th grade, M = 11.7 years old) or the first year of high school (10th grade, M = 15.3 years old) at the outset. Friends resembled one another before the friendship; resemblances were even greater after the friendship began. Resemblances continued to grow among those who remained friends one year later, but declined among those whose friendships dissolved. Partners were not equally responsible for increases in similarity. In stable friendships, the more accepted partner exerted greater influence over the less accepted partner, such that the greatest increases in problem behaviors were found among less accepted youth whose friends had higher initial levels of delinquency and alcohol intoxication. Unstable friends resembled random pairs of youth in that more- and less-accepted partners were comparably uninfluential.
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2. |
- Narusyte, Jurgita, et al.
(author)
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Parental criticism and externalizing behavior problems in adolescents : the role of environment and genotype-environment correlation
- 2011
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In: Journal of Abnormal Psychology. - : American Psychological Association (APA). - 0021-843X .- 1939-1846. ; 120:2, s. 365-376
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Journal article (peer-reviewed)abstract
- Genetic factors are important for the association between parental negativity and child problem behavior, but it is not clear whether this is due to passive or evocative genotype environment correlation (rGE). In this study, we applied the extended children-of-twins model to directly examine the presence of passive and evocative rGE as well as direct environmental effects in the association between parental criticism and adolescent externalizing problem behavior. The cross-sectional data come from the Twin and Offspring Study in Sweden (N = 909 pairs of adult twins) and from the Twin Study of Child and Adolescent Development (N = 915 pairs of twin children). The results revealed that maternal criticism was primarily due to evocative rGE emanating from their adolescent’s externalizing behavior. On the other hand, fathers’ critical remarks tended to affect adolescent problem behavior in a direct environmental way. This suggests that previously reported differences in caretaking between mothers and fathers also are reflected in differences in why parenting is associated with externalizing behavior in offspring.
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3. |
- Patrick, C. J., et al.
(author)
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A construct-network approach to bridging diagnostic and physiological domains : Application to assessment of externalizing psychopathology
- 2013
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In: Journal of Abnormal Psychology. - Washington : American Psychological Association (APA). - 0021-843X .- 1939-1846. ; 122:3, s. 902-916
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Journal article (peer-reviewed)abstract
- A crucial challenge in efforts to link psychological disorders to neural systems, with the aim of developing biologically informed conceptions of such disorders, is the problem of method variance (Campbell & Fiske, 1959). Since even measures of the same construct in differing domains correlate only moderately, it is unsurprising that large sample studies of diagnostic biomarkers yield only modest associations. To address this challenge, a construct-network approach is proposed in which psychometric operationalizations of key neurobehavioral constructs serve as anchors for identifying neural indicators of psychopathology-relevant dispositions, and as vehicles for bridging between domains of clinical problems and neurophysiology. An empirical illustration is provided for the construct of inhibition disinhibition, which is of central relevance to problems entailing deficient impulse control. Findings demonstrate that: (1) a well-designed psychometric index of trait disinhibition effectively predicts externalizing problems of multiple types, (2) this psychometric measure of disinhibition shows reliable brain response correlates, and (3) psychometric and brain-response indicators can be combined to form a joint psychoneurometric factor that predicts effectively across clinical and physiological domains. As a methodology for bridging between clinical problems and neural systems, the construct-network approach provides a concrete means by which existing conceptions of psychological disorders can accommodate and be reshaped by neurobiological insights.
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4. |
- Pennington, Bruce, et al.
(author)
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Individual prediction of dyslexia by single vs. multiple deficit models.
- 2012
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In: Journal of Abnormal Psychology. - : American Psychological Association. - 0021-843X .- 1939-1846. ; 121:1, s. 212-224
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Journal article (peer-reviewed)abstract
- The overall goals of this study were to test single versus multiple cognitive deficit models of dyslexia (reading disability) at the level of individual cases and to determine the clinical utility of these models for prediction and diagnosis of dyslexia. To accomplish these goals, we tested five cognitive models of dyslexia-two single-deficit models, two multiple-deficit models, and one hybrid model-in two large population-based samples, one cross-sectional (Colorado Learning Disability Research Center) and one longitudinal (International longitudinal Twin Study). The cognitive deficits included in these cognitive models were in phonological awareness, language skill, and processing speed and/or naming speed. To determine whether an individual case fit one of these models, we used two methods: 1) the presence or absence of the predicted cognitive deficits, and 2) whether the individuals level of reading skill best fit the regression equation with the relevant cognitive predictors (i.e., whether their reading skill was proportional to those cognitive predictors.) We found that roughly equal proportions of cases met both tests of model fit for the multiple deficit models (30-36%) and single deficit models (24-28%); hence, the hybrid model provided the best overall fit to the data. The remaining roughly 40% of cases in each sample lacked the deficit or deficits that corresponded with their best-fitting regression model. We discuss the clinical implications of these results for both diagnosis of school-age children and preschool prediction of children at risk for dyslexia.
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5. |
- Pisetsky, EM, et al.
(author)
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Suicide attempts in women with eating disorders
- 2013
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In: Journal of abnormal psychology. - : American Psychological Association (APA). - 1939-1846 .- 0021-843X. ; 122:4, s. 1042-1056
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Journal article (peer-reviewed)
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6. |
- Ronald, A., et al.
(author)
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Symptoms of Autism and ADHD: A Swedish Twin Study Examining Their Overlap
- 2014
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In: Journal of Abnormal Psychology. - Washington DC, USA : American Psychological Association (APA). - 0021-843X .- 1939-1846. ; 123:2, s. 440-451
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Journal article (peer-reviewed)abstract
- Autism spectrum disorders (ASD) and attention deficit hyperactivity disorder (ADHD) show high comorbidity. The following questions were addressed regarding their specific symptoms: What is the factor structure of ASD and ADHD symptoms, to what degree do different symptom domains cluster together, to what extent are these domains caused by the same genetic and environmental influences, and what is the best model of their co-occurrence? A population-based twin cohort of over 17,000 9- and 12-year-olds were assessed using the Autism-Tics, AD/HD, and other Comorbidities parental interview inventory. Principal component analyses were conducted, and symptom domain clustering was assessed. Four multivariate twin models were compared. Factors split into three ASD (social impairments, communication impairments, and restricted repetitive behaviors and interests), and three ADHD (inattention, hyperactivity, and impulsivity) symptom domains. Some ASD-ADHD symptom domain combinations clustered together often, although others not at all. A two-factor common pathway model fit the data, suggesting that ASD and ADHD symptom domains tap into separate "ASD" and "ADHD" latent factors that showed high genetic overlap. All subdomains also showed significant specific genetic and environmental influences, reflecting the etiological heterogeneity both within and between ASD and ADHD. These findings support the conceptual distinction of ASD and ADHD, and demonstrate the considerable natural co-occurrence of particular ASD/ADHD symptom domains. The results imply that more children with 1 condition show features of the other condition than show complete comorbidity. Emphasis on symptom co-occurrence, rather than complete comorbidity between disorders, may help focus clinical approaches and advance molecular genetic research.
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