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- Barnes, PD, et al.
(author)
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Antihyperon-hyperon production in the threshold region at LEAR
- 1996
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In: PHYSICS OF ATOMIC NUCLEI. - : AMER INST PHYSICS. ; , s. 1460-1466
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Conference paper (peer-reviewed)abstract
- The experiment PSI185 studies the antihyperon-hyperon production in antiproton-proton annihilation at LEAR/CERN up to 2 GeV/c. This is achieved by investigation of the channels (p) over bar p --> <(Lambda)over bar>Lambda, <(Lambda)over bar>Sigma(0) + c.c.
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2. |
- Johansson, Tord, et al.
(author)
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Results from PS185.
- 1999
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In: NUCLEAR PHYSICS A. - : ELSEVIER SCIENCE BV. ; , s. 173C-178C
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Conference paper (peer-reviewed)abstract
- The PS185 experiment at LEAR/CERN has studied strangeness production in antiproton-proton collisions. Exclusive final states of antihyperon-hyperon pairs have been measured in order to investigate strange-quark production dynamics. A comprehensive data se
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6. |
- Miller, S J, et al.
(author)
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A novel type of regulatory element is required for promoter-specific activity of the PDGF-B intronic enhancer region.
- 1998
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In: Growth Factors. - 0897-7194 .- 1029-2292. ; 16:2, s. 137-51
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Journal article (peer-reviewed)abstract
- We have previously described a non-classical, promoter-specific enhancer for the human Platelet-Derived Growth Factor B (PDGF-B) gene. In JEG-3 choriocarcinoma cells the activity of the enhancer depends upon co-operation with a sequence (the Enhancer-Dependent cis Co-activator "EDC" element) within the promoter. The PDGF-B enhancer fails to activate heterologous promoters, indicating that promoter-specificity depends on an element within the enhancer that can recognise a target sequence within the promoter. Here we identify a sequence within the enhancer of the PDGF-B gene which directs activation of the PDGF-B promoter by distal cis-acting elements. This specifies the wild-type PDGF-B promoter as the target for the enhancer and has been designated the EDC specificity element (EDCse). The cell-type specific nature of this interaction is extended by the observation that the EDCse is also dispensable for enhancer activity in breast-cancer cells (ZR-75). Concomitant to this observation, JEG-3 and ZR-75 cells differ in the binding of nuclear factors to the EDCse. We discuss the relevance of the EDC/EDCse system in regulation of gene expression.
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