| 1. |
- Abazov, V. M., et al.
(author)
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The upgraded DO detector
- 2006
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In: Nuclear Instruments and Methods in Physics Research Section A. - : Elsevier BV. - 0168-9002 .- 1872-9576. ; 565:2, s. 463-537
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Journal article (peer-reviewed)abstract
- The DO experiment enjoyed a very successful data-collection run at the Fermilab Tevatron collider between 1992 and 1996. Since then, the detector has been upgraded to take advantage of improvements to the Tevatron and to enhance its physics capabilities. We describe the new elements of the detector, including the silicon microstrip tracker, central fiber tracker, solenoidal magnet, preshower detectors, forward muon detector, and forward proton detector. The uranium/liquid -argon calorimeters and central muon detector, remaining from Run 1, are discussed briefly. We also present the associated electronics, triggering, and data acquisition systems, along with the design and implementation of software specific to DO.
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| 2. |
- Ablikim, M., et al.
(author)
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Measurements of (XcJ)-> K+K-K+K- decays
- 2006
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In: Physics Letters B. - : Elsevier BV. - 0370-2693 .- 1873-2445. ; 642:3, s. 197-202
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Journal article (peer-reviewed)abstract
- Using 14M psi(2S) events taken with the BESII detector, chi(cJ) -> 2(K+K-) decays are studied. For the four-kaon final state, the branching fractions are B(chi(c0,1,2) ->.2(K+K-)) = (3.48 +/- 0.23 +/- 0.47) x 10(-3), (0.70 +/- 0.13 +/- 0.10) x 10(-3), and (2.17 +/- 0.20 +/- 0.31) x 10(-3). For the phi K+K- final state, the branching fractions, which are measured for the first time, are B(chi(c0,1,2) -> phi K+K-) = (1.03 +/- 0.22 +/- 0.15) x 10(-3), (0.46 +/- 0.16 +/- 0.06) x 10(-3), and (1.67 +/- 0.26 +/- 0.24) x 10(-4). For the phi phi final state, B(chi(c0,2) -> phi phi) = (0.94 +/- 0.21 +/- 0.13) x 10(-3) and (1.70 +/- 0.30 +/- 0.25) x 10(-3).
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| 3. |
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| 4. |
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| 5. |
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| 6. |
- Ballantyne, C., et al.
(author)
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Collaborative meta-analysis of individual participant data from observational studies of Lp-PLA(2) and cardiovascular diseases
- 2007
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In: European Journal of Cardiovascular Prevention & Rehabilitation. - 1741-8275. ; 14:1, s. 41344-41344
-
Research review (peer-reviewed)abstract
- Background A large number of observational epidemiological studies have reported generally positive associations' between circulating mass and activity levels of lipoprotein-associated phospholipase A(2) (Lp-PLA(2)) and the risk of cardiovascular diseases. Few studies have been large enough to provide reliable estimates in different circumstances, such as in different subgroups (e.g., by age group, sex, or smoking status) or at different Lp-PLA2 levels. Moreover, most published studies have related disease risk only to baseline values of Lp-PLA(2) markers (which can lead to substantial underestimation of any risk relationships because of within-person variability over time) and have used different approaches to adjustment for possible confounding factors. Objectives By combination of data from individual participants from all relevant observational studies in a systematic,meta-analysis, with correction for regression dilution (using available data on serial measurements of Lp-PLA(2)), the Lp-PLA(2) Studies Collaboration will aim to characterize more precisely than has previously been possible the strength and shape of the age and sex-specific associations of plasma Lp-PLA(2) with coronary heart disease (and, where data are sufficient with other vascular diseases, such as ischaemic stroke). It will also help to determine to what extent such associations are independent of possible confounding factors and to explore potential sources of heterogeneity among studies, such as those related to assay methods and study design. It is anticipated that the present collaboration will serve as a framework to investigate related questions on Lp-PLA(2) and cardiovascular outcomes. Methods A central database is being established containing data on circulating Lp-PLA(2) values, sex and other potential confounding factors, age at baseline Lp-PLA(2) Measurement, age at event or at last follow-up, major vascular morbidity and cause-specific mortality. Information about any repeat measurements of Lp-PLA2 and potential confounding factors has been sought to allow adjustment for possible confounding and correction for regression dilution. The analyses will involve age-specific regression models. Synthesis of the available observational studies of Lp-PLA(2) will yield information on a total of about 15 000 cardiovascular disease endpoints.
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| 7. |
- Ballantyne, C., et al.
(author)
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Collaborative meta-analysis of individual participant data from observational studies of Lp-PLA2 and cardiovascular diseases
- 2007
-
In: European Journal of Cardiovascular Prevention & Rehabilitation. - : Oxford University Press (OUP). - 1741-8267 .- 1741-8275 .- 2047-4873. ; 14:1, s. 3-11
-
Research review (peer-reviewed)abstract
- BACKGROUND: A large number of observational epidemiological studies have reported generally positive associations between circulating mass and activity levels of lipoprotein-associated phospholipase A2 (Lp-PLA2) and the risk of cardiovascular diseases. Few studies have been large enough to provide reliable estimates in different circumstances, such as in different subgroups (e.g., by age group, sex, or smoking status) or at different Lp-PLA2 levels. Moreover, most published studies have related disease risk only to baseline values of Lp-PLA2 markers (which can lead to substantial underestimation of any risk relationships because of within-person variability over time) and have used different approaches to adjustment for possible confounding factors. OBJECTIVES: By combination of data from individual participants from all relevant observational studies in a systematic 'meta-analysis', with correction for regression dilution (using available data on serial measurements of Lp-PLA2), the Lp-PLA2 Studies Collaboration will aim to characterize more precisely than has previously been possible the strength and shape of the age and sex-specific associations of plasma Lp-PLA2 with coronary heart disease (and, where data are sufficient, with other vascular diseases, such as ischaemic stroke). It will also help to determine to what extent such associations are independent of possible confounding factors and to explore potential sources of heterogeneity among studies, such as those related to assay methods and study design. It is anticipated that the present collaboration will serve as a framework to investigate related questions on Lp-PLA2 and cardiovascular outcomes. METHODS: A central database is being established containing data on circulating Lp-PLA2 values, sex and other potential confounding factors, age at baseline Lp-PLA2 measurement, age at event or at last follow-up, major vascular morbidity and cause-specific mortality. Information about any repeat measurements of Lp-PLA2 and potential confounding factors has been sought to allow adjustment for possible confounding and correction for regression dilution. The analyses will involve age-specific regression models. Synthesis of the available observational studies of Lp-PLA2 will yield information on a total of about 15 000 cardiovascular disease endpoints.
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| 8. |
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| 9. |
- Ma, J, et al.
(author)
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Genetic influences of the intercellular adhesion molecule 1 (ICAM-1) gene polymorphisms in development of Type 1 diabetes and diabetic nephropathy.
- 2006
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In: Diabetic Medicine. - : Wiley. - 0742-3071 .- 1464-5491. ; 23:10, s. 1093-1099
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Journal article (peer-reviewed)abstract
- AIM: The intercellular adhesion molecule-1 (ICAM-1) gene is located on chromosome 19p13, which is linked to Type 1 diabetes (T1D). ICAM-1 expression is related to development of T1D and diabetic nephropathy. The present study aims to evaluate the genetic influence of ICAM-1 gene polymorphisms on the development of T1D and diabetic nephropathy. METHODS: Five valid single nucleotide polymorphisms (SNPs) were genotyped in 432 T1D patients (196 patients had diabetic nephropathy) and 187 non-diabetic control subjects by using dynamic allele-specific hybridization (DASH) and pyrosequencing. RESULTS: SNPs rs281432(C/G) and rs5498 E469K(A/G) had high heterozygous indexes. They were significantly associated with T1D [P = 0.026, OR = 1.644 (95% CI 1.138-2.376) and P < 0.001, OR = 2.456 (1.588-3.8)]. Frequencies of the C allele in SNP rs281432(C/G) and the A allele in SNP rs5498 E469K(A/G) increased stepwise from non-diabetic control subjects to T1D patients without diabetic nephropathy and T1D patients with diabetic nephropathy. Further analysis for these two SNPs indicated that T1D patients had increased frequency of the common haplotype C-A, in comparison with non-diabetic control subjects (38.1 vs. 32.1%, P = 0.035). CONCLUSION: The present study provided evidence that SNPs rs281432(C/G) and rs5498 E469K(A/G) in the ICAM-1 gene confer susceptibility to the development of T1D and might also be associated with diabetic nephropathy in Swedish Caucasians.
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| 10. |
- Sacco, R. L., et al.
(author)
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Aspirin and extended-release dipyridamole versus clopidogrel for recurrent stroke
- 2008
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In: New England Journal of Medicine. - Boston : Massachusetts medical society. - 1533-4406 .- 0028-4793. ; 359:12, s. 1238-51
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Journal article (peer-reviewed)abstract
- BACKGROUND: Recurrent stroke is a frequent, disabling event after ischemic stroke. This study compared the efficacy and safety of two antiplatelet regimens--aspirin plus extended-release dipyridamole (ASA-ERDP) versus clopidogrel. METHODS: In this double-blind, 2-by-2 factorial trial, we randomly assigned patients to receive 25 mg of aspirin plus 200 mg of extended-release dipyridamole twice daily or to receive 75 mg of clopidogrel daily. The primary outcome was first recurrence of stroke. The secondary outcome was a composite of stroke, myocardial infarction, or death from vascular causes. Sequential statistical testing of noninferiority (margin of 1.075), followed by superiority testing, was planned. RESULTS: A total of 20,332 patients were followed for a mean of 2.5 years. Recurrent stroke occurred in 916 patients (9.0%) receiving ASA-ERDP and in 898 patients (8.8%) receiving clopidogrel (hazard ratio, 1.01; 95% confidence interval [CI], 0.92 to 1.11). The secondary outcome occurred in 1333 patients (13.1%) in each group (hazard ratio for ASA-ERDP, 0.99; 95% CI, 0.92 to 1.07). There were more major hemorrhagic events among ASA-ERDP recipients (419 [4.1%]) than among clopidogrel recipients (365 [3.6%]) (hazard ratio, 1.15; 95% CI, 1.00 to 1.32), including intracranial hemorrhage (hazard ratio, 1.42; 95% CI, 1.11 to 1.83). The net risk of recurrent stroke or major hemorrhagic event was similar in the two groups (1194 ASA-ERDP recipients [11.7%], vs. 1156 clopidogrel recipients [11.4%]; hazard ratio, 1.03; 95% CI, 0.95 to 1.11). CONCLUSIONS: The trial did not meet the predefined criteria for noninferiority but showed similar rates of recurrent stroke with ASA-ERDP and with clopidogrel. There is no evidence that either of the two treatments was superior to the other in the prevention of recurrent stroke. (ClinicalTrials.gov number, NCT00153062.)
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| 11. |
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| 12. |
- Wu, Xifeng, et al.
(author)
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Genetic variation in the prostate stem cell antigen gene PSCA confers susceptibility to urinary bladder cancer.
- 2009
-
In: Nature genetics. - : Springer Science and Business Media LLC. - 1546-1718 .- 1061-4036. ; 41:9, s. 991-5
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Journal article (peer-reviewed)abstract
- We conducted a genome-wide association study on 969 bladder cancer cases and 957 controls from Texas. For fast-track validation, we evaluated 60 SNPs in three additional US populations and validated the top SNP in nine European populations. A missense variant (rs2294008) in the PSCA gene showed consistent association with bladder cancer in US and European populations. Combining all subjects (6,667 cases, 39,590 controls), the overall P-value was 2.14 x 10(-10) and the allelic odds ratio was 1.15 (95% confidence interval 1.10-1.20). rs2294008 alters the start codon and is predicted to cause truncation of nine amino acids from the N-terminal signal sequence of the primary PSCA translation product. In vitro reporter gene assay showed that the variant allele significantly reduced promoter activity. Resequencing of the PSCA genomic region showed that rs2294008 is the only common missense SNP in PSCA. Our data identify rs2294008 as a new bladder cancer susceptibility locus.
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| 13. |
- Yusuf, S., et al.
(author)
-
Telmisartan to prevent recurrent stroke and cardiovascular events
- 2008
-
In: New England Journal of Medicine. - : Massachusetts medical society. - 1533-4406 .- 0028-4793. ; 359:12, s. 1225-37
-
Journal article (peer-reviewed)abstract
- BACKGROUND: Prolonged lowering of blood pressure after a stroke reduces the risk of recurrent stroke. In addition, inhibition of the renin-angiotensin system in high-risk patients reduces the rate of subsequent cardiovascular events, including stroke. However, the effect of lowering of blood pressure with a renin-angiotensin system inhibitor soon after a stroke has not been clearly established. We evaluated the effects of therapy with an angiotensin-receptor blocker, telmisartan, initiated early after a stroke. METHODS: In a multicenter trial involving 20,332 patients who recently had an ischemic stroke, we randomly assigned 10,146 to receive telmisartan (80 mg daily) and 10,186 to receive placebo. The primary outcome was recurrent stroke. Secondary outcomes were major cardiovascular events (death from cardiovascular causes, recurrent stroke, myocardial infarction, or new or worsening heart failure) and new-onset diabetes. RESULTS: The median interval from stroke to randomization was 15 days. During a mean follow-up of 2.5 years, the mean blood pressure was 3.8/2.0 mm Hg lower in the telmisartan group than in the placebo group. A total of 880 patients (8.7%) in the telmisartan group and 934 patients (9.2%) in the placebo group had a subsequent stroke (hazard ratio in the telmisartan group, 0.95; 95% confidence interval [CI], 0.86 to 1.04; P=0.23). Major cardiovascular events occurred in 1367 patients (13.5%) in the telmisartan group and 1463 patients (14.4%) in the placebo group (hazard ratio, 0.94; 95% CI, 0.87 to 1.01; P=0.11). New-onset diabetes occurred in 1.7% of the telmisartan group and 2.1% of the placebo group (hazard ratio, 0.82; 95% CI, 0.65 to 1.04; P=0.10). CONCLUSIONS: Therapy with telmisartan initiated soon after an ischemic stroke and continued for 2.5 years did not significantly lower the rate of recurrent stroke, major cardiovascular events, or diabetes. (ClinicalTrials.gov number, NCT00153062.)
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| 14. |
- Axelberg, Peter, et al.
(author)
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A measurement method for determining the direction of propagation of flicker and for tracing a flicker source
- 2005
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In: CIRED. ; , s. 505-508
-
Conference paper (peer-reviewed)abstract
- This paper describes a new and highly efficient measurement method (algorithm) that determines how flicker propagates throughout the network and also traces the dominant flicker source. The fundamental principle of the method is to use the fact that a flicker source produces an amplitude modulation in the voltage and current waveforms. The low frequency variations in voltage and current that cause flicker are retrieved in a demodulation and filtering process. By first multiplying the low frequency variations in voltage and current and then integrate, a new quantity, flicker power, is achieved. The sign and the magnitude of flicker power give the direction to the flicker source as well as tracing the dominating flicker source.
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| 15. |
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| 16. |
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| 17. |
- Axelberg, Peter G.V. 1959, et al.
(author)
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AUTOMATIC CLASSIFICATION OF VOLTAGE EVENTS USING THE SUPPORT VECTOR MACHINE METHOD
- 2007
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In: 19th International Conference on Electricity Distribution (SIRED 2007) , Vienna, Austria, 21-24 May, 2007.
-
Conference paper (peer-reviewed)abstract
- Statistically based classification systems need to be trained on a large number of training data in order to classify unseen data accurately. However, it is difficult to gather enough voltage events for the training purpose from real recordings. Therefore, a classification system trained to accurately classify real voltage events, but based on synthetic training data is highly in demand. This paper therefore proposes the design of a statistically based classification system trained on synthetic data. The paper gives also the results of conducted performance tests when the proposed classification system was trained to classify seven common types of voltage events. The experiments showed an overall detection rate of 81.6%, 91.9% and 99.5% respectively.
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| 18. |
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| 19. |
- Axelberg, Peter G.V., et al.
(author)
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Performance Tests of a Support Vector Machine used for Classification of Voltage Disturbances
- 2006
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In: in proc. of 12th International conf. on Harmonics and Quality of Power (ICHQP 2006), Cascais, Portugal, Oct.1-5, 2006.
-
Conference paper (peer-reviewed)abstract
- This paper proposes a novel method for classifying voltage disturbances in electric power systems by using the Support Vector Machine (SVM) method. The proposed SVM classifier is designed to classify five common types of voltage disturbances and experiments have been conducted on recorded disturbances with good classification results. The proposed SVM classifier is also shown to be robust in terms of using training data and testing data that originate from two different power networks.
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| 20. |
- Axelberg, P.G.V., et al.
(author)
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Support vector machine for classification of voltage disturbances
- 2007
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In: IEEE Transactions on Power Delivery. - : IEEE. - 0885-8977 .- 1937-4208. ; 22:3, s. 1297-1303
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Journal article (peer-reviewed)abstract
- The support vector machine (SVM) is a powerful method for statistical classification of data used in a number of different applications. However, the usefulness of the method in a commercial available system is very much dependent on whether the SVM classifier can be pretrained from a factory since it is not realistic that the SVM classifier must be trained by the customers themselves before it can be used. This paper proposes a novel SVM classification system for voltage disturbances. The performance of the proposed SVM classifier is investigated when the voltage disturbance data used for training and testing originated from different sources. The data used in the experiments were obtained from both real disturbances recorded in two different power networks and from synthetic data. The experimental results shown high accuracy in classification with training data from one power network and unseen testing data from another. High accuracy was also achieved when the SVM classifier was trained on data from a real power network and test data originated from synthetic data. A lower accuracy resulted when the SVM classifier was trained on synthetic data and test data originated from the power network.
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| 21. |
- Axelberg, Peter G.V. 1959, et al.
(author)
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Support Vector Machine for Classification of Voltage Disturbances
- 2007
-
In: accepted for publication in IEEE Transactions on Power Delivery. ; 22:3, s. 1297-1303, July, 2007
-
Journal article (peer-reviewed)abstract
- The Support Vector Machine (SVM) is a powerful method for statistical classification of data used in a number of different applications. However, the usefulness of the method in a commercial available system is very much dependent on whether the SVM classifier can be pre-trained from a factory since it is not realistic that the SVM classifier must be trained by the customers themselves before it can be used. We first propose a novel SVM classification system for voltage disturbances. Our aim also includes investigating the performance of the proposed SVM classifier when the voltage disturbance data used for training and testing are originated from different sources. The data used in the experiments were originated from both real disturbances recorded in two different power networks and from synthetic data. The experimental results have shown excellent accuracy in classification when training data were originated from one power network and unseen testing data from another. High accuracy was also achieved when the SVM classifier was trained on data from a real power network and test data originated from synthetic data. Slightly less accuracy was achieved when the SVM classifier was trained on synthetic data and test data were originated from the power network.
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| 22. |
- Axelberg, Peter G.V. 1959, et al.
(author)
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Trace of flicker sources by using the quantity of flicker power
- 2007
-
In: IEEE transactions on Power Delivery. ; 23:1, s. pp.465-471
-
Journal article (peer-reviewed)abstract
- Industries that produce flicker are often placed close to each other and connected to the same power grid system. This implies that the measured flicker level at the point of common coupling (PCC) is a result of contribution from a number of different flicker sources. In a mitigation process it is essential to know which one of the flicker sources is the dominant one. We propose a method to determine the flicker propagations and trace the flicker sources by using flicker power measurements. Flicker power is considered as a quantity containing both sign and magnitude. The sign determines if a flicker source is placed downstream or upstream with respect to a given monitoring point and the magnitude is used to determine the propagation of flicker power throughout the power network and to trace the dominant flicker source. This paper covers the theoretical background of flicker power and describes a novel method for calculation of flicker power that can be implemented in a power network analyzer. Also conducted simulations and a field test based on the proposed method will be described in the paper.
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| 23. |
- Axelberg, P., et al.
(author)
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Trace of Flicker Sources by using the Quantity of Flicker power.
- 2008
-
In: IEEE Transactions on Power Delivery. - : IEEE. - 0885-8977 .- 1937-4208. ; 23:1, s. 465-471
-
Journal article (peer-reviewed)abstract
- Industries that produce flicker are often placed close to each other and connected to the same power grid system. This implies that the measured flicker level at the point of common coupling (PCC) is a result of contribution from a number of different flicker sources. In a mitigation process it is essential to know which one of the flicker sources is the dominant one. We propose a method to determine the flicker propagations and trace the flicker sources by using flicker power measurements. Flicker power is considered as a quantity containing both sign and magnitude. The sign determines if a flicker source is placed downstream or upstream with respect to a given monitoring point and the magnitude is used to determine the propagation of flicker power throughout the power network and to trace the dominant flicker source. This paper covers the theoretical background of flicker power and describes a novel method for calculation of flicker power that can be implemented in a power network analyzer. Also conducted simulations and a field test based on the proposed method will be described in the paper.
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| 24. |
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| 25. |
- Bollen, Math, et al.
(author)
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Bridging the gap between signal and power
- 2009
-
In: IEEE signal processing magazine (Print). - 1053-5888 .- 1558-0792. ; 26:4, s. 12-31
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Journal article (peer-reviewed)abstract
- Signal processing has been used in many different applications, including electric power systems. This is an important category, since a wide variety of digital measurements is available and data analysis is required to deliver diagnostic solutions and correlation with known behaviors. Measurements are taken at numerous locations, and the analysis of data applies to a variety of issues in ¿ power quality (PQ) and reliability ¿ power system and equipment diagnostics ¿ power system control ¿ power system protection. This article focuses on problems and issues related to PQ and power system diagnostics, in particular those where signal processing techniques are extremely important. PQ is a general term that describes the quality of voltage and current waveforms. PQ problems include all electric power problems or disturbances in the supply system that prevent end-user equipment from operating properly. Examples of voltage and current variations that can result in PQ problems include voltage interruptions, long- and short-duration voltage variations, steady-state research opportunities that use the measured voltages and currents to indicate possible equipment and system problems (referred to as equipment diagnostics).
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| 26. |
- Bollen, Math, et al.
(author)
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Characterization of voltage variations in the very-short time-scale
- 2005
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In: IEEE Transactions on Power Delivery. - 0885-8977 .- 1937-4208. ; 20:2, s. 1198-1199
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Journal article (peer-reviewed)abstract
- This letter presents a method for characterizing the fast voltage variations that occur on a time scale between the subsecond fluctuations covered by the flickermeter standard and the 10-min values covered by standards like EN 50160 The method is fully compatible with IEC 61000-4-30 class A. The new characteristic is correlated to small switching actions, such as domestic load switching and transformer tap-changer operation.
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| 27. |
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| 28. |
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| 29. |
- Bollen, Math H.J., et al.
(author)
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On the Analysis of Voltage and Current Transients in Three-Phase Power Systems
- 2007
-
In: IEEE Transactions on Power Delivery. ; 22:2, s. 1194-1201, April 2007
-
Journal article (peer-reviewed)abstract
- This paper presents a method for analysis measurementsof voltage transients in three-phase systems. The method is based on the Clarke transform introduced in 1950 forcalculations of travelling waves along three-phase transmission lines. The proposed method also shows close similarities with the classification of three-phase unbalanced voltage dips into types C and D. After extracting the actual transient, e.g. by using a notch filter centered on the power-system frequency, the three signals are decomposed into seven components. From the relationbetween these seven components, the dominant component isidentified. The method is successfully applied to a number of measured transients. The paper also identifies the limitations of the method and gives suggestions for future work.
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| 30. |
- Bollen, Math H. J., et al.
(author)
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Signal processing of power-quality disturbances
- 2006
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In: Johh Wiley & Sons - IEEE Press. - Piscataway, NJ : IEEE Communications Society. - 0471731684 ; , s. 888-
-
Book (pop. science, debate, etc.)abstract
- Bridging the gap between power quality and signal processing This innovative new text brings together two leading experts, one from signal processing and the other from power quality. Combining their fields of expertise, they set forth and investigate various types of power quality disturbances, how measurements of these disturbances are processed and interpreted, and, finally, the use and interpretation of power quality standards documents. As a practical aid to readers, the authors make a clear distinction between two types of power quality disturbances: Variations: disturbances that are continuously present Events: disturbances that occur occasionally A complete analysis and full set of tools are provided for each type of disturbance: Detailed examination of the origin of the disturbance Signal processing measurement techniques, including advanced techniques and those techniques set forth in standards documents Interpretation and analysis of measurement data Methods for further processing the features extracted from the signal processing into site and system indices The depth of coverage is outstanding: the authors present and analyze material that is not covered in the standards nor found in the scientific literature. This text is intended for two groups of readers: students and researchers in power engineering who need to use signal processing techniques for power system applications, and students and researchers in signal processing who need to perform power system disturbance analyses and diagnostics. It is also highly recommended for any engineer or utility professional involved in power quality monitoring.
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| 31. |
- Bollen, Math, et al.
(author)
-
On the analysis of voltage and current transients in three-phase power systems
- 2007
-
In: IEEE Transactions on Power Delivery. - 0885-8977 .- 1937-4208. ; 22:2, s. 1194-1201
-
Journal article (peer-reviewed)abstract
- This paper proposes a method for analyzing measurements of voltage transients in three-phase systems. The method is based on the Clarke transform introduced in 1950 for calculations of travelling waves along three-phase transmission lines. The proposed method also shows close similarities with the classification of three-phase unbalanced voltage dips. After extracting the actual transient (e.g., by using a notch filter centered on the power-system frequency), the three signals are decomposed into seven components. From the relation between these seven components, the dominant component is identified. The method is successfully applied to a number of measured transients. The paper also identifies the limitations of the method and gives suggestions for future work
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| 32. |
- Bruzzi, M, et al.
(author)
-
Radiation-hard semiconductor detectors for SuperLHC
- 2005
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In: Nuclear Instruments & Methods in Physics Research. Section A: Accelerators, Spectrometers, Detectors, and Associated Equipment. - : Elsevier BV. - 0167-5087 .- 0168-9002. ; 541:1-2, s. 189-201
-
Journal article (peer-reviewed)abstract
- An option of increasing the luminosity of the Large Hadron Collider (LHC) at CERN to 1035 cm-2 s-1 has been envisaged to extend the physics reach of the machine. An efficient tracking down to a few centimetres from the interaction point will be required to exploit the physics potential of the upgraded LHC. As a consequence, the semiconductor detectors close to the interaction region will receive severe doses of fast hadron irradiation and the inner tracker detectors will need to survive fast hadron fluences of up to above 1016cm-2. The CERN-RD50 project "Development of Radiation Hard Semiconductor Devices for Very High Luminosity Colliders" has been established in 2002 to explore detector materials and technologies that will allow to operate devices up to, or beyond, this limit. The strategies followed by RD50 to enhance the radiation tolerance include the development of new or defect engineered detector materials (SiC, GaN, Czochralski and epitaxial silicon, oxygen enriched Float Zone silicon), the improvement of present detector designs and the understanding of the microscopic defects causing the degradation of the irradiated detectors. The latest advancements within the RD50 collaboration on radiation hard semiconductor detectors will be reviewed and discussed in this work.
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| 33. |
- Diener, Hans-Christoph, et al.
(author)
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Effects of aspirin plus extended-release dipyridamole versus clopidogrel and telmisartan on disability and cognitive function after recurrent stroke in patients with ischaemic stroke in the Prevention Regimen for Effectively Avoiding Second Strokes (PRoFESS) trial : a double-blind, active and placebo-controlled study.
- 2008
-
In: Lancet Neurology. - : Elsevier. - 1474-4422 .- 1474-4465. ; 7:10, s. 875-884
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Journal article (peer-reviewed)abstract
- BACKGROUND: The treatment of ischaemic stroke with neuroprotective drugs has been unsuccessful, and whether these compounds can be used to reduce disability after recurrent stroke is unknown. The putative neuroprotective effects of antiplatelet compounds and the angiotensin II receptor antagonist telmisartan were investigated in the Prevention Regimen for Effectively Avoiding Second Strokes (PRoFESS) trial.METHODS: Patients who had had an ischaemic stroke were randomly assigned in a two by two factorial design to receive either 25 mg aspirin (ASA) and 200 mg extended-release dipyridamole (ER-DP) twice a day or 75 mg clopidogrel once a day, and either 80 mg telmisartan or placebo once per day. The predefined endpoints for this substudy were disability after a recurrent stroke, assessed with the modified Rankin scale (mRS) and Barthel index at 3 months, and cognitive function, assessed with the mini-mental state examination (MMSE) score at 4 weeks after randomisation and at the penultimate visit. Analysis was by intention to treat. The study was registered with ClinicalTrials.gov, number NCT00153062.FINDINGS: 20,332 patients (mean age 66 years) were randomised and followed-up for a median of 2.4 years. Recurrent strokes occurred in 916 (9%) patients randomly assigned to ASA with ER-DP and 898 (9%) patients randomly assigned to clopidogrel; 880 (9%) patients randomly assigned to telmisartan and 934 (9%) patients given placebo had recurrent strokes. mRS scores were not statistically different in patients with recurrent stroke who were treated with ASA and ER-DP versus clopidogrel (p=0.38), or with telmisartan versus placebo (p=0.61). There was no significant difference in the proportion of patients with recurrent stroke with a good outcome, as measured with the Barthel index, across all treatment groups. Additionally, there was no significant difference in the median MMSE scores, the percentage of patients with an MMSE score of 24 points or less, the percentage of patients with a drop in MMSE score of 3 points or more between 1 month and the penultimate visit, and the number of patients with dementia among the treatment groups. There were no significant differences in the proportion of patients with cognitive impairment or dementia among the treatment groups.INTERPRETATION: Disability due to recurrent stroke and cognitive decline in patients with ischaemic stroke were not different between the two antiplatelet regimens and were not affected by the preventive use of telmisartan.
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| 34. |
- Feng, S, et al.
(author)
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3D Face Recognition Using Affine Integral Invariants
- 2006
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In: IEEE international conf. on ASSP (ICASSP-06). ; II, s. 189-192
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Conference paper (peer-reviewed)abstract
- A new 3D face representation and recognition approach is presented in this paper. Two sets of facial curves are extracted from a face range image, and a novel facial feature representation, the affine integral invariant, is introduced to mitigate the effect of pose on the facial curves. A human face is shown to be representable by a smallsubset of those affine integral invariant curves. A recognition procedure based on the Discriminant Analysis and Jensen-Shannon Divergence analysis is proposed. Substantiating examples are provided with an achieved classification accuracy of 92.57%.
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| 35. |
- Gu, G, et al.
(author)
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Estrogen protects primary osteocytes against glucocorticoid-induced apoptosis
- 2005
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In: Apoptosis. - : Springer Science and Business Media LLC. - 1360-8185 .- 1573-675X. ; 10:3, s. 583-595
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Journal article (peer-reviewed)abstract
- Glucocorticoid-induced osteoporosis may be at least in part due to the increased apoptosis of osteocytes. To study the role of osteocyte apoptosis in glucocorticoid-induced osteoporosis, we isolated primary osteocytes from murine calvaria for the analysis of the effects of dexamethasone in in vitro culture. The cells were identified by morphology, cytochemical staining, immunocytochemical staining and mRNA expression of phosphate-regulating gene with homology to endopeptidases on the X chromosome (PHEX) and sclerosteosis/van Buchem disease gene (SOST). We found that dexamethasone induced osteocyte apoptosis in a dose-dependent manner. A glucocorticoid receptor antagonist, mifepristone (RU486), suppressed dexamethasone-Induced osteocyte apoptosis, suggesting that it was mediated by glucocorticoid receptor. Immunocytochemical stainings showed that glucocorticoid receptors are present in primary osteocytes, and they were translocated to nuclei after the exposure to dexamethasone. Addition of estrogen prevented glucocorticoid receptor translocation into nuclei. Corresponding antiapoptotic effects in primary osteocytes were also seen after the pretreatment of primary osteocytes with a picomolar concentration of estrogen. The pure antiestrogen ICI 182,780 inhibited estrogen effect on apoptosis induced by dexamethasone. These data suggest that glucocorticoid receptors play an important role in glucocorticoid-induced osteocyte apoptosis. Most importantly, estrogen has a protective effect against osteocyte apoptosis. To conclude, the mechanism of glucocorticoid-induced osteoporosis may be due to the apoptosis of osteocytes, which can be opposed by estrogen.
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| 37. |
- Lavebratt, C., et al.
(author)
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Association study between chromosome 10q26.11 and obesity among Swedish men
- 2005
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In: International Journal of Obesity. - : Springer Science and Business Media LLC. - 0307-0565 .- 1476-5497. ; 29:12, s. 1422-1428
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Journal article (peer-reviewed)abstract
- OBJECTIVE: Proximal chromosome 10q26 was recently linked to waist/hip ratio in European and African-American families. The objective was to investigate whether genomic variation in chromosome 10q26.11 reflects variation in obesity-related clinical parameters in a Swedish population. DESIGN: Genetic association study of single-nucleotide polymorphisms (SNPs) in chromosome 10q26.11 and obesity-related clinical parameters was performed. Obesity was defined as body mass index (BMI)≥30 kg/m2. SUBJECTS: Swedish Caucasians comprising 276 obese and 480 nonobese men, 313 obese and 494 nonobese women, 177 obese and 163 nonobese patients with type 2 diabetes mellitus (T2DM), and 106 obese and 201 nonobese subjects with impaired glucose tolerance (IGT) patients. MEASUREMENTS: Genotypes of 11 SNPs at chromosome 10q26.11, and various obesity-related clinical parameters. RESULTS: Homozygosity of a common haplotype constructed by three SNPs, rs2185937, rs1797 and hCV1402327, covering an interval of 2.7 kb, was suggested to confer an increased risk for obesity of 1.5 among men (P=0.043). The C allele frequency and homozygous genotype frequency of the rs1797 tended to be higher among obese compared to among nonobese men (P=0.017 and 0.020, respectively). The distribution of BMI and diastolic blood pressure was higher among those with the C/C genotype (P=0.022 and 0.0061, respectively). The obese and the nonobese groups were homogeneous over BMI subgroups with regard to rs1797 risk genotype distribution. There was no tendency for association between rs1797 and obesity among neither women nor T2DM nor IGT patients. CONCLUSION: We show support for association between proximal chromosome 10q26.11 and obesity among Swedish men but not women through the analysis of a haplotype encompassing 2.7 kb. © 2005 Nature Publishing Group All rights reserved.
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| 42. |
- Wang, Shu Min, 1963, et al.
(author)
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Metamorphic InGaAs quantum wells for light emission at 1.3-1.6 μm
- 2007
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In: Thin Solid Films. - : Elsevier BV. - 0040-6090 .- 1879-2731. ; 515:10, s. 4348-4351
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Journal article (peer-reviewed)abstract
- Metamorphic InGaAs quantum well structures grown on GaAs reveal strong light emission at 1.3-1.6 μm, smooth surface with an average roughness below 2 nm and good rectifying I-V characteristics. Dark line defects are found in the QW. Post growth thermal annealing further improves the luminescence efficiency but does not remove those dark line defects. Some challenges of epitaxial growth using this method for laser applications are discussed. © 2006 Elsevier B.V. All rights reserved.
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