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CTLA4-Ig but not an...
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Ali, Abukar,1988Gothenburg University,Göteborgs universitet,Institutionen för medicin, avdelningen för reumatologi och inflammationsforskning,Institute of Medicine, Department of Rheumatology and Inflammation Research
(author)
CTLA4-Ig but not anti-TNF therapy promotes staphylococcal septic arthritis in mice.
- Article/chapterEnglish2015
Publisher, publication year, extent ...
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2015-04-02
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Oxford University Press (OUP),2015
Numbers
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LIBRIS-ID:oai:gup.ub.gu.se/214831
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https://gup.ub.gu.se/publication/214831URI
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https://doi.org/10.1093/infdis/jiv212DOI
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Subject category:art swepub-publicationtype
Notes
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The development of biologics has greatly increased the quality of life as well as the life expectancy of many RA patients. However, a large number of these patients are at an increased risk of developing serious infections. The aim of this study was to examine differential effects of anti-TNF versus CTLA4-Ig treatment on both immunological response and host defense in a murine model of septic arthritis.
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Welin, Amanda,1983Gothenburg University,Göteborgs universitet,Institutionen för medicin, avdelningen för reumatologi och inflammationsforskning,Institute of Medicine, Department of Rheumatology and Inflammation Research(Swepub:gu)xwelia
(author)
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Schwarze, Jan-ChristophGothenburg University,Göteborgs universitet,Institutionen för medicin, avdelningen för reumatologi och inflammationsforskning,Institute of Medicine, Department of Rheumatology and Inflammation Research
(author)
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Svensson, Mattias,1982Gothenburg University,Göteborgs universitet,Institutionen för medicin, avdelningen för reumatologi och inflammationsforskning,Institute of Medicine, Department of Rheumatology and Inflammation Research(Swepub:gu)xsvmat
(author)
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Na, ManliGothenburg University,Göteborgs universitet,Institutionen för medicin, avdelningen för reumatologi och inflammationsforskning,Institute of Medicine, Department of Rheumatology and Inflammation Research(Swepub:gu)xnamaz
(author)
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Jarneborn, AndersGothenburg University,Göteborgs universitet,Institutionen för medicin,Institute of Medicine(Swepub:gu)xjarna
(author)
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Magnusson, MalinGothenburg University,Göteborgs universitet,Institutionen för medicin,Institute of Medicine
(author)
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Mohammad, MajdGothenburg University,Göteborgs universitet,Institutionen för medicin,Institute of Medicine(Swepub:gu)xmomaj
(author)
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Kwiecinski, Jakub,1985Gothenburg University,Göteborgs universitet,Institutionen för medicin, avdelningen för reumatologi och inflammationsforskning,Institute of Medicine, Department of Rheumatology and Inflammation Research(Swepub:gu)xkwija
(author)
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Josefsson, Elisabet,1966Gothenburg University,Göteborgs universitet,Institutionen för medicin, avdelningen för reumatologi och inflammationsforskning,Institute of Medicine, Department of Rheumatology and Inflammation Research(Swepub:gu)xjosel
(author)
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Bylund, Johan,1975Gothenburg University,Göteborgs universitet,Institutionen för medicin, avdelningen för reumatologi och inflammationsforskning,Institutionen för odontologi, sektion 3,Institute of Medicine, Department of Rheumatology and Inflammation Research,Institute of Odontology, Section 3(Swepub:gu)xbyljo
(author)
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Pullerits, Rille,1969Gothenburg University,Göteborgs universitet,Institutionen för medicin, avdelningen för reumatologi och inflammationsforskning,Institute of Medicine, Department of Rheumatology and Inflammation Research(Swepub:gu)xpulri
(author)
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Jin, Tao,1973Gothenburg University,Göteborgs universitet,Institutionen för medicin, avdelningen för reumatologi och inflammationsforskning,Institute of Medicine, Department of Rheumatology and Inflammation Research(Swepub:gu)xjinta
(author)
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Göteborgs universitetInstitutionen för medicin, avdelningen för reumatologi och inflammationsforskning
(creator_code:org_t)
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In:The Journal of infectious diseases: Oxford University Press (OUP)212:8, s. 1308-13161537-66130022-1899
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