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LIBRIS Formathandbok  (Information om MARC21)
FältnamnIndikatorerMetadata
00004728naa a2200541 4500
001oai:gup.ub.gu.se/318763
003SwePub
008240910s2022 | |||||||||||000 ||eng|
0247 a https://gup.ub.gu.se/publication/3187632 URI
0247 a https://doi.org/10.1177/036354652211158282 DOI
040 a (SwePub)gu
041 a eng
042 9 SwePub
072 7a ref2 swepub-contenttype
072 7a art2 swepub-publicationtype
1001 a Bedrin, M. D.4 aut
2451 0a Prospective Evaluation of Posterior Glenoid Bone Loss After First-time and Recurrent Posterior Glenohumeral Instability Events
264 c 2022-08-19
264 1b SAGE Publications,c 2022
520 a Background: Although posterior glenohumeral instability is becoming an increasingly recognized cause of shoulder pain, the role of posterior glenoid bone loss on outcomes remains incompletely understood. Purposes: To prospectively determine the amount of bone loss associated with posterior instability events and to determine predisposing factors based on preinstability imaging. Study Design: Cross-sectional study; Level of evidence, 3. Methods: A total of 1428 shoulders were evaluated prospectively for >= 4 years. At baseline, a subjective history of shoulder instability was ascertained for each patient, and bilateral noncontrast magnetic resonance imaging (MRI) scans of the shoulders were obtained regardless of any reported history of shoulder instability. The cohort was prospectively followed during the study period, and those who were diagnosed with posterior glenohumeral instability were identified. Postinjury MRI scans were obtained and compared with the screening MRI scans. Glenoid version, perfect-circle-based bone loss was measured for each patient's pre- and postinjury MRI scans using previously described methods. Results: Of the 1428 shoulders that were prospectively followed, 10 shoulders sustained a first-time posterior instability event and 3 shoulders sustained a recurrent posterior instability event. At baseline, 11 of 13 shoulders had some amount of glenoid dysplasia and/or bone loss. The change in glenoid bone loss was 5.4% along the axis of greatest loss (95% CI, 3.8%-7.0%; P = .009), 4.4% at the glenoid equator (95% CI, 2.7%-6.2%; P = .016), and 4.2% of total glenoid area (95% CI, 2.9%-5.3%; P = .002). Recurrent glenoid instability was associated with a greater amount of absolute bone loss along the axis of greatest loss compared with first-time instability (recurrent: 16.8% +/- 1.1%; 95% CI, 14.6%-18.9%; first-time: 10.0% +/- 1.5%; 95% CI, 7.0%-13.0%; P = .005). Baseline glenoid retroversion >= 10 degrees was associated with a significantly greater percentage of bone loss along the axis of greatest loss (>= 10 degrees of retroversion: 13.5% +/- 2.0%; 95% CI, 9.6%-17.4%; <10 degrees of retroversion: 8.5% +/- 0.8%; 95% CI, 7.0%-10.0%; P = .045). Conclusions: Posterior glenohumeral instability events were associated with glenoid bone loss of 5%. The amount of glenoid bone loss after a recurrent posterior glenohumeral instability event was greater than that after first-time instability. Glenoid retroversion >= 10 degrees was associated with a greater amount of posterior glenoid bone loss after a posterior instability event.
650 7a MEDICIN OCH HÄLSOVETENSKAPx Klinisk medicinx Ortopedi0 (SwePub)302112 hsv//swe
650 7a MEDICAL AND HEALTH SCIENCESx Clinical Medicinex Orthopaedics0 (SwePub)302112 hsv//eng
653 a posterior glenohumeral instability
653 a posterior glenoid bone loss
653 a shoulder instability
653 a humeral head
653 a risk-factors
653 a version
653 a epidemiology
653 a tomography
653 a history
653 a repair
653 a ct
653 a Orthopedics
653 a Sport Sciences
7001 a Owens, B. D.4 aut
7001 a Slaven, S. E.4 aut
7001 a LeClere, L. E.4 aut
7001 a Donohue, M. A.4 aut
7001 a Tennent, D. J.4 aut
7001 a Goodlett, R. P.4 aut
7001 a Cameron, K. L.4 aut
7001 a Posner, M. A.4 aut
7001 a Dickens, Jonathanu Gothenburg University,Göteborgs universitet,Institutionen för kliniska vetenskaper, Avdelningen för ortopedi,Institute of Clinical Sciences, Department of Orthopaedics4 aut0 (Swepub:gu)xdicjo
7102 a Göteborgs universitetb Institutionen för kliniska vetenskaper, Avdelningen för ortopedi4 org
7730 t American Journal of Sports Medicined : SAGE Publicationsg 50:11, s. 3028-3035q 50:11<3028-3035x 0363-5465x 1552-3365
8564 8u https://gup.ub.gu.se/publication/318763
8564 8u https://doi.org/10.1177/03635465221115828

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