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Cyclodextrin promotes atherosclerosis regression via macrophage reprogramming

Zimmer, S (author)
Grebe, A (author)
Bakke, SS (author)
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Bode, N (author)
Halvorsen, B (author)
Ulas, T (author)
Skjelland, M (author)
De Nardo, D (author)
Labzin, LI (author)
Kerksiek, A (author)
Hempel, C (author)
Heneka, MT (author)
Hawxhurst, V (author)
Fitzgerald, ML (author)
Trebicka, J (author)
Bjorkhem, I (author)
Karolinska Institutet
Gustafsson, JA (author)
Karolinska Institutet
Westerterp, M (author)
Tall, AR (author)
Wright, SD (author)
Espevik, T (author)
Schultze, JL (author)
Nickenig, G (author)
Lutjohann, D (author)
Latz, E (author)
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 (creator_code:org_t)
American Association for the Advancement of Science (AAAS), 2016
2016
English.
In: Science translational medicine. - : American Association for the Advancement of Science (AAAS). - 1946-6242 .- 1946-6234. ; 8:333, s. 333ra50-
  • Journal article (peer-reviewed)
Abstract Subject headings
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  • The cyclic oligosaccharide 2-hydroxypropyl-β-cyclodextrin facilitates atheroprotective mechanisms through oxysterol-mediated reprogramming of macrophages.

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