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Sökning: WFRF:(Peltonen Lasse)

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  • Jalkanen, Jukka-Pekka, et al. (författare)
  • Modeling of ships as a source of underwater noise
  • 2017
  • Konferensbidrag (övrigt vetenskapligt/konstnärligt)abstract
    • Shipping as a source of underwater noise is increasing. Underwater noise emission levels are seldom considered as a ship design parameter unless low vessel noise is specifically required, like in the case of warships and research vessels. Noise emissions are not regulated yet, but the awareness of possible impacts of noise on marine life is increasing. This paper describes the implementation of a noise source model for the Ship Traffic Emission Abatement Model (STEAM; Jalkanen et al. 2009; 2012; Johansson et al, 2017). The combination of vessel technical description and activity can be used to generate noise source maps which are based on actual ship traffic data. The generated noise source maps can ve used to describe the energy emitted as noise to the water, which facilitates regular annual updates of the noise emissions from ship traffic in the Baltic Sea area.
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4.
  • Peden, John F., et al. (författare)
  • A genome-wide association study in Europeans and South Asians identifies five new loci for coronary artery disease
  • 2011
  • Ingår i: Nature Genetics. - : Springer Science and Business Media LLC. - 1061-4036 .- 1546-1718. ; 43:4, s. 339-344
  • Tidskriftsartikel (refereegranskat)abstract
    • Genome-wide association studies have identified 11 common variants convincingly associated with coronary artery disease (CAD)(1-7), a modest number considering the apparent heritability of CAD(8). All of these variants have been discovered in European populations. We report a meta-analysis of four large genome-wide association studies of CAD, with similar to 575,000 genotyped SNPs in a discovery dataset comprising 15,420 individuals with CAD (cases) (8,424 Europeans and 6,996 South Asians) and 15,062 controls. There was little evidence for ancestry-specific associations, supporting the use of combined analyses. Replication in an independent sample of 21,408 cases and 19,185 controls identified five loci newly associated with CAD (P < 5 x 10(-8) in the combined discovery and replication analysis): LIPA on 10q23, PDGFD on 11q22, ADAMTS7-MORF4L1 on 15q25, a gene rich locus on 7q22 and KIAA1462 on 10p11. The CAD-associated SNP in the PDGFD locus showed tissue-specific cis expression quantitative trait locus effects. These findings implicate new pathways for CAD susceptibility.
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  • Resultat 1-4 av 4
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