| 1. |
- Norell Bergendahl, Margareta, et al.
(author)
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Så skapar du det innovativa företaget
- 2008
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In: Innovationsförmåga. - Stockholm : PIEp. - 9789197785204 ; , s. 18-39
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Book chapter (pop. science, debate, etc.)
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| 2. |
- Olsson, Annika, et al.
(author)
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Ett inspirerande år
- 2008
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In: Innovationsförmåga. - 9789197785204 ; , s. 6-8
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Book chapter (pop. science, debate, etc.)
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| 3. |
- Anderson, Helén, et al.
(author)
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Skapa kundnärvaro i innovationsprocessen
- 2008
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In: Innovationsförmåga. - Malmö : Holmbergs i Malmö AB. - 9789197785204 ; , s. 40-59
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Book chapter (pop. science, debate, etc.)
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| 4. |
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| 5. |
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Litteraturens historia i Sverige
- 2009
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Editorial collection (other academic/artistic)abstract
- Den femte reviderade upplagan av Litteraturens historia i Sverige (red. Olsson & Algulin mfl) tar litteraturen in i 2000-talet, och läsaren möter bland mycket annat den nya arbetarlitteraturen, chick lit och den svenska deckarboomen. Litterära ikoner som Heliga Birgitta, Selma Lagerlöf och August Strindberg har fått sällskap av samtida författare som Jonas Hassen Khemiri, Henning Mankell och Sara Stridsberg
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| 6. |
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| 7. |
- Beckeman, Märit, et al.
(author)
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Driving forces for food packaging development in Sweden - a historical perspective
- 2005
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In: World of Food Science.
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Journal article (peer-reviewed)abstract
- Availability of safe, edible food is essential. Food shortage has caused many wars and encouraged people to try to preserve food for a longer time, allowing storage and transport, often in some kind of package/container. The USA showed the way in developing the food sector and already prior to the 2nd World War they had self-service stores filled with packed goods. Europe, however, had to wait to apply the US ideas until after the war. As Sweden did not take part, the country got a head start, although with restricted imports and exports and rationing of some food. When self-service retail and new food products, particularly frozen food, became available in the 1950s, the role of packaging and distribution became important, providing mass-produced food at reasonable prices. The country took the lead in frozen food in Europe and became the origin of some quite successful packaging companies and distribution/equipment suppliers, starting up as local supporting companies to the food sector but then expanding into Europe in order to grow. The purpose of this study is to identify the key drivers in society, in the food industry and elsewhere that have affected the development of food-retail packaging, with Sweden as an example of a lead country in Europe for some years after WW II. The study is qualitative and based on semi-structured interviews with experts in the field. The results clearly indicate that frozen food technology and the introduction of self-service stores at about the same time and mainly later chilled/fresh food are the three developments that are considered to have had the most profound influence on the food sector development. Packages were considered as a consequence of the appearance of frozen or chilled food and self-service stores by the interviewees and not as very important in itself, even though neither frozen/chilled food nor self-service stores would exist without packaging and efficient distribution.
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| 8. |
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| 9. |
- Berg, Anna-Karin, et al.
(author)
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Antiviral Treatment of Coxsackie B Virus Infection in Human Pancreatic Islets
- 2007
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In: Antiviral Research. - : Elsevier BV. - 0166-3542 .- 1872-9096. ; 74:1, s. 65-71
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Journal article (peer-reviewed)abstract
- Enterovirus infections of the pancreatic islets are believed to trigger or precipitate the near total destruction of β-cells that constitutes type 1 diabetes (T1D). This study investigated the ability of an anti-picornaviral compound, pleconaril, to block the replication of two β-cell tropic Coxsackie B4 virus (CBV-4) strains in isolated human islets. The two strains, VD2921 and V89 4557, with demonstrated abilities to cause non-lytic persistence or lytic infection, respectively, in islets, represented two different potential mechanisms behind virus-induced T1D. The virus replication in the islets was studied with and without addition of pleconaril. In addition, islet morphology was studied every day. To test the effects of pleconaril and/or DMSO on the β-cells’ insulin secretion, glucose perifusions were performed on treated and untreated islets. Virus titrations showed a clear reduction of the replication of both strains after pleconaril treatment. The VD2921 strain was inhibited to undetectable levels. The V89 4557 strain, however, showed an initial reduction of titers but virus titers then increased despite the addition of a second dose of pleconaril. This incomplete inhibition of viral replication suggested the existence of a resistant subtype within this strain. Pleconaril treatment reduced the β-cells’ insulin secretion in response to glucose stimulation in some experiments and induced slight morphological changes to the islets compared to untreated controls. In summary, pleconaril reduced the replication of the two β-cell tropic CBV-4 strains in human islets. However, genetic differences between these strains influenced the effectiveness of pleconaril treatment. This stresses the importance of using multiple viral strains in antiviral tests.
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| 10. |
- Bergquist, Annika, et al.
(author)
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Increased risk of primary sclerosing cholangitis and ulcerative colitis in first-degree relatives of patients with primary sclerosing cholangitis
- 2008
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In: Clinical Gastroenterology and Hepatology. - New York : Elsevier. - 1542-3565 .- 1542-7714. ; 6:8, s. 939-943
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Journal article (peer-reviewed)abstract
- Background & Aims: The importance of genetic factors for the development of primary sclerosing cholangitis (PSC) is incompletely understood. This study assessed the risk of PSC and inflammatory bowel disease (IBD) among first-degree relatives of patients with PSC, compared with the first-degree relatives of a cohort without PSC. Methods: Subjects from the national Swedish cohort of PSC patients (n = 678) were matched for date of birth, sex, and region to up to 10 subjects without a diagnosis of PSC (n = 6347). Linkage through general population registers identified first-degree relatives of subjects in both the PSC and comparison cohorts (n = 34,092). Diagnoses among first-degree relatives were identified by using the Inpatient Register. Results: The risk of cholangitis was statistically significantly increased in offspring, siblings, and parents of the PSC patient cohort, compared with relatives of the comparison cohort, with the hazard ratios and 95% confidence intervals, 11.5 (1.6–84.4), 11.1 (3.3–37.8), and 2.3 (0.9–6.1), respectively. The hazard ratios for ulcerative colitis (UC) among first-degree relatives of all PSC patients was 3.3 (2.3–4.9) and for Crohn's disease 1.4 (0.8–2.5). The risk of UC for relatives of PSC patients without IBD was also increased, 7.4 (2.9–18.9). Conclusions: First-degree relatives of patients with PSC run an increased risk of PSC, indicating the importance of genetic factors in the etiology of PSC. First-degree relatives of PSC patients without IBD are also at an increased risk of UC, which might indicate shared genetic susceptibility factors for PSC and UC.
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