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Genetics and crime :
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Wertz, J.Department of Psychology & Neuroscience, Duke University, Durham, United States
(author)
Genetics and crime : Integrating new genomic discoveries into psychological research about antisocial behavior
- Article/chapterEnglish2018
Publisher, publication year, extent ...
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2018-03-07
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New York :Cambridge University Press,2018
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printrdacarrier
Numbers
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LIBRIS-ID:oai:DiVA.org:oru-113805
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https://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-113805URI
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https://doi.org/10.1177/0956797617744542DOI
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https://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-156599URI
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Language:English
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Summary in:English
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Subject category:ref swepub-contenttype
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Subject category:art swepub-publicationtype
Notes
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The E-Risk Study is funded by UK Medical Research Council (MRC) Grant G1002190. The Dunedin Multidisciplinary Health and Development Research Unit is funded by the New Zealand Health Research Council and the New Zealand Ministry of Business, Innovation and Employment. This research was supported by National Institute on Aging Grant AG032282, National Institute of Child Health and Human Development Grant HD077482, MRC Grant MR/P005918/1, the Jacobs Foundation, and the Avielle Foundation. Data support was provided by Duke’s Social Science Research Institute and North Carolina Biotechnology Center Grant 2016-IDG-1013. D. W. Belsky and C. L. Odgers are supported by fellowships from the Jacobs Foundation. A. L. Beckley is supported by a Forte Marie Curie International Fellowship.
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Drawing on psychological and sociological theories of crime causation, we tested the hypothesis that genetic risk for low educational attainment (assessed via a genome-wide polygenic score) is associated with criminal offending. We further tested hypotheses of how polygenic risk relates to the development of antisocial behavior from childhood through adulthood. Across the Dunedin and Environmental Risk (E-Risk) birth cohorts of individuals growing up 20 years and 20,000 kilometers apart, education polygenic scores predicted risk of a criminal record with modest effects. Polygenic risk manifested during primary schooling in lower cognitive abilities, lower self-control, academic difficulties, and truancy, and it was associated with a life-course-persistent pattern of antisocial behavior that onsets in childhood and persists into adulthood. Crime is central in the nature-nurture debate, and findings reported here demonstrate how molecular-genetic discoveries can be incorporated into established theories of antisocial behavior. They also suggest that improving school experiences might prevent genetic influences on crime from unfolding.
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Caspi, A.Department of Psychology & Neuroscience, Duke University, Durham, United States; Department of Psychiatry & Behavioral Sciences, Duke University School of Medicine, Durham, United States; Center for Genomic and Computational Biology, Duke University, Durham, United States; Social, Genetic, & Developmental Psychiatry Research Centre, Institute of Psychiatry, Psychology, & Neuroscience, King's College London, London, England
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Belsky, D. W.Department of Medicine, Duke University, School of Medicine, Durham, United States; Social Science Research Institute, Duke University, Durham, United States
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Beckley, Amber,1981-Stockholms universitet,Sociologiska institutionen,Duke University, USA,Department of Psychology & Neuroscience, Duke University, Durham, United States; Demography Unit, Department of Sociology, Stockholm University, Stockholm, Sweden(Swepub:su)ambe0256
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Arseneault, L.Social, Genetic, & Developmental Psychiatry Research Centre, Institute of Psychiatry, Psychology, & Neuroscience, King's College London, London, England
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Barnes, J. C.School of Criminal Justice, University of Cincinnati, Ohio, United States
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Corcoran, D. L.Center for Genomic and Computational Biology, Duke University, Durham, United States
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Hogan, S.Dunedin Multidisciplinary Health and Development Research Unit, Department of Psychology, University of Otago, Dunedin, New Zealand
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Houts, R. M.Department of Psychology & Neuroscience, Duke University, Durham, United States
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Morgan, N.Home Office, London, United Kingdom
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Odgers, C. L.Sanford School of Public Policy, Duke University, Durham, United States
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Prinz, J. A.Center for Genomic and Computational Biology, Duke University, Durham, United States
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Sugden, K.Department of Psychology & Neuroscience, Duke University, Durham, United States
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Williams, B. S.Department of Psychology & Neuroscience, Duke University, Durham, United States
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Poulton, R.Dunedin Multidisciplinary Health and Development Research Unit, Department of Psychology, University of Otago, Dunedin, New Zealand
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Moffitt, T. E.Department of Psychology & Neuroscience, Duke University, Durham, United States; Department of Psychiatry & Behavioral Sciences, Duke University School of Medicine, Durham, United States; Center for Genomic and Computational Biology, Duke University, Durham, United States; Social, Genetic, & Developmental Psychiatry Research Centre, Institute of Psychiatry, Psychology, & Neuroscience, King's College, London, England
(author)
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Department of Psychology & Neuroscience, Duke University, Durham, United StatesDepartment of Psychology & Neuroscience, Duke University, Durham, United States; Department of Psychiatry & Behavioral Sciences, Duke University School of Medicine, Durham, United States; Center for Genomic and Computational Biology, Duke University, Durham, United States; Social, Genetic, & Developmental Psychiatry Research Centre, Institute of Psychiatry, Psychology, & Neuroscience, King's College London, London, England
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In:Psychological ScienceNew York : Cambridge University Press29:5, s. 791-8030956-79761467-9280
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Wertz, J.
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Caspi, A.
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Belsky, D. W.
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Beckley, Amber, ...
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Arseneault, L.
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Barnes, J. C.
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Corcoran, D. L.
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Hogan, S.
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Houts, R. M.
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Morgan, N.
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Odgers, C. L.
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Prinz, J. A.
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Sugden, K.
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Williams, B. S.
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Poulton, R.
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Moffitt, T. E.
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