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Sökning: id:"swepub:oai:DiVA.org:kth-313309" > Athero-occlusive Di...

LIBRIS Formathandbok  (Information om MARC21)
FältnamnIndikatorerMetadata
00005301naa a2200433 4500
001oai:DiVA.org:kth-313309
003SwePub
008220602s2022 | |||||||||||000 ||eng|
0247 a https://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-3133092 URI
0247 a https://doi.org/10.1016/j.ejvs.2021.12.0382 DOI
040 a (SwePub)kth
041 a engb eng
042 9 SwePub
072 7a ref2 swepub-contenttype
072 7a art2 swepub-publicationtype
1001 a Matthews, Evan O.u James Cook Univ, Coll Med & Dent, Queensland Res Ctr Peripheral Vasc Dis, Townsville, Qld 4811, Australia.4 aut
2451 0a Athero-occlusive Disease Appears to be Associated with Slower Abdominal Aortic Aneurysm Growth :b An Exploratory Analysis of the TEDY Trial
264 1b Elsevier BV,c 2022
338 a print2 rdacarrier
500 a QC 20220602
520 a Objective: The role of atherosclerosis in abdominal aortic aneurysm (AAA) pathogenesis is controversial. The aim of this study was to compare AAA growth in patients who did and did not have concurrent athero-occlusive disease (AOD). Methods: Patients with an AAA measuring 35 - 49 mm in maximum diameter were recruited as part of the TElmisartan in the management of abdominal aortic aneurysm (TEDY) trial. TEDY participants who had infrarenal aortic volume and orthogonal diameter assessed by computed tomography at entry and at least one other time point during the trial (12 and/or 24 months) were included. AOD was defined by prior diagnoses of coronary heart disease, stroke, or peripheral arterial disease or an ankle brachial pressure index < 0.90. The increase in AAA volume and diameter from entry for participants who did and did not have AOD was assessed using linear mixed effects models; 131 of the 210 participants recruited to TEDY were included. Results: In an unadjusted analysis, the mean (95% confidence interval) annual increases in AAA volume and diameter for participants with AOD were 3.26 (0.82 - 5.70) cm(3) and 0.70 (0.19 - 1.22) mm slower than those without AOD, p = .008 and.007 respectively. The association between AOD and significantly slower AAA growth was maintained after adjusting for risk factors and medications, significantly unequally distributed between participants with and without an AOD diagnosis. Conclusion: In an exploratory analysis of a selective cohort from the TEDY trial, AOD was associated with slower AAA growth. Validation of these findings in other cohorts is needed.
650 7a MEDICIN OCH HÄLSOVETENSKAPx Klinisk medicinx Kardiologi0 (SwePub)302062 hsv//swe
650 7a MEDICAL AND HEALTH SCIENCESx Clinical Medicinex Cardiac and Cardiovascular Systems0 (SwePub)302062 hsv//eng
653 a Abdominal aortic aneurysm
653 a Athero-occlusive disease
653 a Growth
7001 a Moxon, Joseph, Vu James Cook Univ, Coll Med & Dent, Queensland Res Ctr Peripheral Vasc Dis, Townsville, Qld 4811, Australia.;James Cook Univ, Ctr Mol Therapeut, Australian Inst Trop Hlth & Med, Townsville, Qld, Australia.4 aut
7001 a Singh, Tejas P.u James Cook Univ, Coll Med & Dent, Queensland Res Ctr Peripheral Vasc Dis, Townsville, Qld 4811, Australia.;Townsville Univ Hosp, Dept Vasc & Endovasc Surg, Townsville, Qld, Australia.4 aut
7001 a Thanigaimani, Shivshankaru James Cook Univ, Coll Med & Dent, Queensland Res Ctr Peripheral Vasc Dis, Townsville, Qld 4811, Australia.;James Cook Univ, Ctr Mol Therapeut, Australian Inst Trop Hlth & Med, Townsville, Qld, Australia.4 aut
7001 a Jones, Rhondda E.u James Cook Univ, Coll Med & Dent, Queensland Res Ctr Peripheral Vasc Dis, Townsville, Qld 4811, Australia.;James Cook Univ, Ctr Mol Therapeut, Australian Inst Trop Hlth & Med, Townsville, Qld, Australia.4 aut
7001 a Gasser, Thomas C.u KTH,Hållfasthetslära4 aut0 (Swepub:kth)u1dvxi4s
7001 a Fitridge, Robertu Univ Adelaide, Discipline Surg, Adelaide, SA, Australia.;Royal Adelaide Hosp, Dept Vasc & Endovasc Surg, Cent Adelaide Local Hlth Network, Adelaide, SA, Australia.;Queen Elizabeth Hosp, Div Surg, Woodville South, SA, Australia.4 aut
7001 a Lindeman, Jan H. N.u Leiden Univ, Dept Vasc Surg, Med Ctr, Leiden, Netherlands.4 aut
7001 a Dalman, Ronald L.u Stanford Univ, Sch Med, Dept Surg, Stanford, CA 94305 USA.;Vet Affairs Palo Alto Hlth Care Syst, Palo Alto, CA USA.4 aut
7001 a Golledge, Jonathanu James Cook Univ, Coll Med & Dent, Queensland Res Ctr Peripheral Vasc Dis, Townsville, Qld 4811, Australia.;James Cook Univ, Ctr Mol Therapeut, Australian Inst Trop Hlth & Med, Townsville, Qld, Australia.;Townsville Univ Hosp, Dept Vasc & Endovasc Surg, Townsville, Qld, Australia.4 aut
7102 a James Cook Univ, Coll Med & Dent, Queensland Res Ctr Peripheral Vasc Dis, Townsville, Qld 4811, Australia.b James Cook Univ, Coll Med & Dent, Queensland Res Ctr Peripheral Vasc Dis, Townsville, Qld 4811, Australia.;James Cook Univ, Ctr Mol Therapeut, Australian Inst Trop Hlth & Med, Townsville, Qld, Australia.4 org
7730 t European Journal of Vascular and Endovascular Surgeryd : Elsevier BVg 63:4, s. 632-640q 63:4<632-640x 1078-5884x 1532-2165
8564 8u https://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-313309
8564 8u https://doi.org/10.1016/j.ejvs.2021.12.038

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