Sökning: id:"swepub:oai:DiVA.org:uu-52572" > Enhanced splicing o...
Fältnamn | Indikatorer | Metadata |
---|---|---|
000 | 01911naa a2200277 4500 | |
001 | oai:DiVA.org:uu-52572 | |
003 | SwePub | |
008 | 081017s1995 | |||||||||||000 ||eng| | |
024 | 7 | a https://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-525722 URI |
040 | a (SwePub)uu | |
041 | a engb eng | |
042 | 9 SwePub | |
072 | 7 | a ref2 swepub-contenttype |
072 | 7 | a art2 swepub-publicationtype |
100 | 1 | a Mühlemann, Oliveru Uppsala universitet,Institutionen för medicinsk biokemi och mikrobiologi4 aut |
245 | 1 0 | a Enhanced splicing of nonconsensus 3' splice sites late during adenovirus infection |
264 | 1 | c 1995 |
338 | a print2 rdacarrier | |
520 | a The adenovirus major late transcription unit is an example of an alternatively spliced gene, in which a common 5' splice site can be spliced to more than 15 different 3' splice sites. Here we show that the specificity in 3' splice site recognition changes during virus infection, such that 3' splice sites with long consensus-type polypyrimidine tracts are repressed while 3' splice sites with short atypical polypyrimidine tracts, which bind U2AF65K inefficiently, are enhanced in splicing in late virus-infected nuclear extracts. On the basis of these experiments, we discuss a mechanism that helps to explain how the complex pattern of major late mRNAs is produced late during virus infection. | |
653 | a MEDICINE | |
653 | a MEDICIN | |
700 | 1 | a Kreivi, Jan-Peteru Uppsala universitet,Institutionen för medicinsk biokemi och mikrobiologi4 aut |
700 | 1 | a Akusjärvi, Göranu Uppsala universitet,Institutionen för medicinsk biokemi och mikrobiologi4 aut0 (Swepub:uu)gak30132 |
710 | 2 | a Uppsala universitetb Institutionen för medicinsk biokemi och mikrobiologi4 org |
773 | 0 | t Journal of Virologyg 69:11, s. 7324-7327q 69:11<7324-7327x 0022-538Xx 1098-5514 |
856 | 4 8 | u https://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-52572 |
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