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FältnamnIndikatorerMetadata
00006344naa a2200985 4500
001oai:gup.ub.gu.se/302389
003SwePub
008240528s2021 | |||||||||||000 ||eng|
009oai:DiVA.org:umu-176354
009oai:DiVA.org:uu-430697
0247 a https://gup.ub.gu.se/publication/3023892 URI
0247 a https://doi.org/10.1016/j.envint.2020.1062192 DOI
0247 a https://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1763542 URI
0247 a https://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-4306972 URI
040 a (SwePub)gud (SwePub)umud (SwePub)uu
041 a eng
042 9 SwePub
072 7a ref2 swepub-contenttype
072 7a art2 swepub-publicationtype
1001 a Nordeide Kuiper, I.4 aut
2451 0a Lifelong exposure to air pollution and greenness in relation to asthma, rhinitis and lung function in adulthood
264 1b Elsevier BV,c 2021
520 a Objectives: To investigate if air pollution and greenness exposure from birth till adulthood affects adult asthma, rhinitis and lung function. Methods: We analysed data from 3428 participants (mean age 28) in the RHINESSA study in Norway and Sweden. Individual mean annual residential exposures to nitrogen dioxide (NO2), particulate matter (PM10 and PM2.5), black carbon (BC), ozone (O3) and greenness (normalized difference vegetation index (NDVI)) were averaged across susceptibility windows (0–10 years, 10–18 years, lifetime, adulthood (year before study participation)) and analysed in relation to physician diagnosed asthma (ever/allergic/non-allergic), asthma attack last 12 months, current rhinitis and low lung function (lower limit of normal (LLN), z-scores of forced expiratory volume in one second (FEV1), forced vital capacity (FVC) and FEV1/FVC below 1.64). We performed logistic regression for asthma attack, rhinitis and LLN lung function (clustered with family and study centre), and conditional logistic regression with a matched case-control design for ever/allergic/non-allergic asthma. Multivariable models were adjusted for parental asthma and education. Results: Childhood, adolescence and adult exposure to NO2, PM10 and O3 were associated with an increased risk of asthma attacks (ORs between 1.29 and 2.25), but not with physician diagnosed asthma. For rhinitis, adulthood exposures seemed to be most important. Childhood and adolescence exposures to PM2.5 and O3 were associated with lower lung function, in particular FEV1 (range ORs 2.65 to 4.21). No associations between NDVI and asthma or rhinitis were revealed, but increased NDVI was associated with lower FEV1 and FVC in all susceptibility windows (range ORs 1.39 to 1.74). Conclusions: Air pollution exposures in childhood, adolescence and adulthood were associated with increased risk of asthma attacks, rhinitis and low lung function in adulthood. Greenness was not associated with asthma or rhinitis, but was a risk factor for low lung function. © 2020 The Authors
650 7a MEDICIN OCH HÄLSOVETENSKAPx Hälsovetenskapx Arbetsmedicin och miljömedicin0 (SwePub)303032 hsv//swe
650 7a MEDICAL AND HEALTH SCIENCESx Health Sciencesx Occupational Health and Environmental Health0 (SwePub)303032 hsv//eng
650 7a MEDICIN OCH HÄLSOVETENSKAPx Klinisk medicinx Lungmedicin och allergi0 (SwePub)302192 hsv//swe
650 7a MEDICAL AND HEALTH SCIENCESx Clinical Medicinex Respiratory Medicine and Allergy0 (SwePub)302192 hsv//eng
653 a Air pollution
653 a Asthma
653 a Greenness
653 a Lung function
653 a Rhinitis
653 a Biological organs
653 a Logistic regression
653 a Nitrogen oxides
653 a Air pollution exposures
653 a Conditional logistic regressions
653 a Multi-variable models
653 a Nitrogen dioxides
653 a Normalized difference vegetation index
653 a Particulate Matter
653 a PM10 and PM2.5
653 a Diseases
653 a adolescence
653 a adult
653 a allergy
653 a atmospheric pollution
653 a health risk
653 a multivariate analysis
653 a pollution effect
653 a pollution exposure
653 a public health
653 a risk factor
653 a Norway
653 a Sweden
653 a Air pollution
7001 a Svanes, C.4 aut
7001 a Markevych, I.4 aut
7001 a Accordini, S.4 aut
7001 a Bertelsen, R. J.4 aut
7001 a Bråbäck, Lennartu Umeå universitet,Avdelningen för hållbar hälsa4 aut0 (Swepub:umu)lebr0005
7001 a Heile Christensen, J.4 aut
7001 a Forsberg, Bertilu Umeå universitet,Avdelningen för hållbar hälsa4 aut0 (Swepub:umu)befo0001
7001 a Halvorsen, T.4 aut
7001 a Heinrich, J.4 aut
7001 a Hertel, O.4 aut
7001 a Hoek, G.4 aut
7001 a Holm, Mathias,d 1969u Gothenburg University,Göteborgs universitet,Institutionen för medicin, avdelningen för samhällsmedicin och folkhälsa,Institute of Medicine, School of Public Health and Community Medicine4 aut0 (Swepub:gu)xhomat
7001 a de Hoogh, K.4 aut
7001 a Janson, Christeru Uppsala universitet,Lung- allergi- och sömnforskning4 aut0 (Swepub:uu)chrisjn
7001 a Malinovschi, Andrei,d 1978-u Uppsala universitet,Klinisk fysiologi4 aut0 (Swepub:uu)anmal535
7001 a Marcon, A.4 aut
7001 a Miodini Nilsen, R.4 aut
7001 a Sigsgaard, T.4 aut
7001 a Johannessen, A.4 aut
7102 a Umeå universitetb Avdelningen för hållbar hälsa4 org
7730 t Environmental Internationald : Elsevier BVg 146q 146x 0160-4120x 1873-6750
8564 u https://doi.org/10.1016/j.envint.2020.106219
8564 u https://doi.org/10.1016/j.envint.2020.106219y Fulltext
8564 u https://umu.diva-portal.org/smash/get/diva2:1485262/FULLTEXT01.pdfx primaryx Raw objecty fulltext:print
8564 u https://uu.diva-portal.org/smash/get/diva2:1516436/FULLTEXT01.pdfx primaryx Raw objecty fulltext:print
8564 8u https://gup.ub.gu.se/publication/302389
8564 8u https://doi.org/10.1016/j.envint.2020.106219
8564 8u https://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-176354
8564 8u https://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-430697

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