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FältnamnIndikatorerMetadata
00004266naa a2200589 4500
001oai:gup.ub.gu.se/177213
003SwePub
008240528s2013 | |||||||||||000 ||eng|
009oai:prod.swepub.kib.ki.se:126860964
024a https://gup.ub.gu.se/publication/1772132 URI
024a https://doi.org/10.1038/bjc.2013.2012 DOI
024a http://kipublications.ki.se/Default.aspx?queryparsed=id:1268609642 URI
040 a (SwePub)gud (SwePub)ki
041 a eng
042 9 SwePub
072 7a ref2 swepub-contenttype
072 7a art2 swepub-publicationtype
100a Andersen, T V4 aut
2451 0a Patterns of exposure to infectious diseases and social contacts in early life and risk of brain tumours in children and adolescents: an International Case-Control Study (CEFALO).
264 c 2013-05-07
264 1b Springer Science and Business Media LLC,c 2013
520 a Background:Infectious diseases and social contacts in early life have been proposed to modulate brain tumour risk during late childhood and adolescence.Methods:CEFALO is an interview-based case-control study in Denmark, Norway, Sweden and Switzerland, including children and adolescents aged 7-19 years with primary intracranial brain tumours diagnosed between 2004 and 2008 and matched population controls.Results:The study included 352 cases (participation rate: 83%) and 646 controls (71%). There was no association with various measures of social contacts: daycare attendance, number of childhours at daycare, attending baby groups, birth order or living with other children.Cases of glioma and embryonal tumours had more frequent sick days with infections in the first 6 years of life compared with controls. In 7-19 year olds with 4+ monthly sick day, the respective odds ratios were 2.93 (95% confidence interval: 1.57-5.50) and 4.21 (95% confidence interval: 1.24-14.30).Interpretation:There was little support for the hypothesis that social contacts influence childhood and adolescent brain tumour risk. The association between reported sick days due to infections and risk of glioma and embryonal tumour may reflect involvement of immune functions, recall bias or inverse causality and deserve further attention.British Journal of Cancer advance online publication 7 May 2013; doi:10.1038/bjc.2013.201 www.bjcancer.com.
650 7a MEDICIN OCH HÄLSOVETENSKAPx Klinisk medicinx Pediatrik0 (SwePub)302212 hsv//swe
650 7a MEDICAL AND HEALTH SCIENCESx Clinical Medicinex Pediatrics0 (SwePub)302212 hsv//eng
650 7a MEDICIN OCH HÄLSOVETENSKAPx Klinisk medicinx Cancer och onkologi0 (SwePub)302032 hsv//swe
650 7a MEDICAL AND HEALTH SCIENCESx Clinical Medicinex Cancer and Oncology0 (SwePub)302032 hsv//eng
650 7a MEDICIN OCH HÄLSOVETENSKAPx Klinisk medicinx Infektionsmedicin0 (SwePub)302092 hsv//swe
650 7a MEDICAL AND HEALTH SCIENCESx Clinical Medicinex Infectious Medicine0 (SwePub)302092 hsv//eng
653 a childhood brain tumour
653 a infectious disease
653 a social contact
653 a epidemiology
653 a children
700a Schmidt, L S4 aut
700a Poulsen, A H4 aut
700a Feychting, Mu Karolinska Institutet4 aut
700a Röösli, M4 aut
700a Tynes, T4 aut
700a Aydin, D4 aut
700a Prochazka, M4 aut
700a Lannering, Birgitta,d 1948u Gothenburg University,Göteborgs universitet,Institutionen för kliniska vetenskaper, Avdelningen för pediatrik,Institute of Clinical Sciences, Department of Pediatrics4 aut0 (Swepub:gu)xlabir
700a Klæboe, L4 aut
700a Eggen, T4 aut
700a Kuehni, C E4 aut
700a Schmiegelow, K4 aut
700a Schüz, J4 aut
710a Karolinska Institutetb Institutionen för kliniska vetenskaper, Avdelningen för pediatrik4 org
773t British journal of cancerd : Springer Science and Business Media LLCg 108, s. 2346-2353q 108<2346-2353x 1532-1827x 0007-0920
856u https://www.nature.com/articles/bjc2013201.pdf
8564 8u https://gup.ub.gu.se/publication/177213
8564 8u https://doi.org/10.1038/bjc.2013.201
8564 8u http://kipublications.ki.se/Default.aspx?queryparsed=id:126860964

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