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Sökning: WFRF:(Ma Ying) > (2010-2014) > Meta-analysis of ge...

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FältnamnIndikatorerMetadata
00004364naa a2201045 4500
001oai:DiVA.org:uu-275696
003SwePub
008160204s2012 | |||||||||||000 ||eng|
024a https://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-2756962 URI
024a https://doi.org/10.1038/ng.10192 DOI
040 a (SwePub)uu
041 a engb eng
042 9 SwePub
072 7a ref2 swepub-contenttype
072 7a art2 swepub-publicationtype
100a Cho, Yoon Shin4 aut
2451 0a Meta-analysis of genome-wide association studies identifies eight new loci for type 2 diabetes in east Asians.
264 c 2011-12-11
264 1b Springer Science and Business Media LLC,c 2012
338 a print2 rdacarrier
520 a We conducted a three-stage genetic study to identify susceptibility loci for type 2 diabetes (T2D) in east Asian populations. We followed our stage 1 meta-analysis of eight T2D genome-wide association studies (6,952 cases with T2D and 11,865 controls) with a stage 2 in silico replication analysis (5,843 cases and 4,574 controls) and a stage 3 de novo replication analysis (12,284 cases and 13,172 controls). The combined analysis identified eight new T2D loci reaching genome-wide significance, which mapped in or near GLIS3, PEPD, FITM2-R3HDML-HNF4A, KCNK16, MAEA, GCC1-PAX4, PSMD6 and ZFAND3. GLIS3, which is involved in pancreatic beta cell development and insulin gene expression, is known for its association with fasting glucose levels. The evidence of an association with T2D for PEPD and HNF4A has been shown in previous studies. KCNK16 may regulate glucose-dependent insulin secretion in the pancreas. These findings, derived from an east Asian population, provide new perspectives on the etiology of T2D.
700a Chen, Chien-Hsiun4 aut
700a Hu, Cheng4 aut
700a Long, Jirong4 aut
700a Ong, Rick Twee Hee4 aut
700a Sim, Xueling4 aut
700a Takeuchi, Fumihiko4 aut
700a Wu, Ying4 aut
700a Go, Min Jin4 aut
700a Yamauchi, Toshimasa4 aut
700a Chang, Yi-Cheng4 aut
700a Kwak, Soo Heon4 aut
700a Ma, Ronald C W4 aut
700a Yamamoto, Ken4 aut
700a Adair, Linda S4 aut
700a Aung, Tin4 aut
700a Cai, Qiuyin4 aut
700a Chang, Li-Ching4 aut
700a Chen, Yuan-Tsong4 aut
700a Gao, Yutang4 aut
700a Hu, Frank B4 aut
700a Kim, Hyung-Lae4 aut
700a Kim, Sangsoo4 aut
700a Kim, Young Jin4 aut
700a Lee, Jeannette Jen-Mai4 aut
700a Lee, Nanette R4 aut
700a Li, Yun4 aut
700a Liu, Jian Jun4 aut
700a Lu, Wei4 aut
700a Nakamura, Jiro4 aut
700a Nakashima, Eitaro4 aut
700a Ng, Daniel Peng-Keat4 aut
700a Tay, Wan Ting4 aut
700a Tsai, Fuu-Jen4 aut
700a Wong, Tien Yin4 aut
700a Yokota, Mitsuhiro4 aut
700a Zheng, Wei4 aut
700a Zhang, Rong4 aut
700a Wang, Congrong4 aut
700a So, Wing Yee4 aut
700a Ohnaka, Keizo4 aut
700a Ikegami, Hiroshi4 aut
700a Hara, Kazuo4 aut
700a Cho, Young Min4 aut
700a Cho, Nam H4 aut
700a Chang, Tien-Jyun4 aut
700a Bao, Yuqian4 aut
700a Hedman, Åsa K4 aut
700a Morris, Andrew P4 aut
700a McCarthy, Mark I4 aut
700a Takayanagi, Ryoichi4 aut
700a Park, Kyong Soo4 aut
700a Jia, Weiping4 aut
700a Chuang, Lee-Ming4 aut
700a Chan, Juliana C N4 aut
700a Maeda, Shiro4 aut
700a Kadowaki, Takashi4 aut
700a Lee, Jong-Young4 aut
700a Wu, Jer-Yuarn4 aut
700a Teo, Yik Ying4 aut
700a Tai, E Shyong4 aut
700a Shu, Xiao Ou4 aut
700a Mohlke, Karen L4 aut
700a Kato, Norihiro4 aut
700a Han, Bok-Ghee4 aut
700a Seielstad, Mark4 aut
773t Nature Geneticsd : Springer Science and Business Media LLCg 44:1q 44:1x 1061-4036x 1546-1718
856u https://europepmc.org/articles/pmc3582398?pdf=render
8564 8u https://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-275696
8564 8u https://doi.org/10.1038/ng.1019

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