WFRF:(Mattingsdal M.)
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| Fältnamn | Indikatorer | Metadata |
|---|---|---|
| 000 | 17266naa a2204729 4500 | |
| 001 | oai:gup.ub.gu.se/290860 | |
| 003 | SwePub | |
| 008 | 240528s2021 | |||||||||||000 ||eng| | |
| 009 | oai:prod.swepub.kib.ki.se:143007973 | |
| 024 | 7 | a https://gup.ub.gu.se/publication/2908602 URI |
| 024 | 7 | a https://doi.org/10.1111/adb.128802 DOI |
| 024 | 7 | a http://kipublications.ki.se/Default.aspx?queryparsed=id:1430079732 URI |
| 040 | a (SwePub)gud (SwePub)ki | |
| 041 | a eng | |
| 042 | 9 SwePub | |
| 072 | 7 | a ref2 swepub-contenttype |
| 072 | 7 | a art2 swepub-publicationtype |
| 100 | 1 | a Munn-Chernoff, M. A.4 aut |
| 245 | 1 0 | a Shared genetic risk between eating disorder- and substance-use-related phenotypes: Evidence from genome-wide association studies |
| 264 | c 2020-02-16 | |
| 264 | 1 | b Wiley,c 2021 |
| 520 | a Eating disorders and substance use disorders frequently co-occur. Twin studies reveal shared genetic variance between liabilities to eating disorders and substance use, with the strongest associations between symptoms of bulimia nervosa and problem alcohol use (genetic correlation [r(g)], twin-based = 0.23-0.53). We estimated the genetic correlation between eating disorder and substance use and disorder phenotypes using data from genome-wide association studies (GWAS). Four eating disorder phenotypes (anorexia nervosa [AN], AN with binge eating, AN without binge eating, and a bulimia nervosa factor score), and eight substance-use-related phenotypes (drinks per week, alcohol use disorder [AUD], smoking initiation, current smoking, cigarettes per day, nicotine dependence, cannabis initiation, and cannabis use disorder) from eight studies were included. Significant genetic correlations were adjusted for variants associated with major depressive disorder and schizophrenia. Total study sample sizes per phenotype ranged from similar to 2400 to similar to 537 000 individuals. We used linkage disequilibrium score regression to calculate single nucleotide polymorphism-based genetic correlations between eating disorder- and substance-use-related phenotypes. Significant positive genetic associations emerged between AUD and AN (r(g) = 0.18; false discovery rate q = 0.0006), cannabis initiation and AN (r(g) = 0.23; q < 0.0001), and cannabis initiation and AN with binge eating (r(g) = 0.27; q = 0.0016). Conversely, significant negative genetic correlations were observed between three nondiagnostic smoking phenotypes (smoking initiation, current smoking, and cigarettes per day) and AN without binge eating (r(gs) = -0.19 to -0.23; qs < 0.04). The genetic correlation between AUD and AN was no longer significant after co-varying for major depressive disorder loci. The patterns of association between eating disorder- and substance-use-related phenotypes highlights the potentially complex and substance-specific relationships among these behaviors. | |
| 650 | 7 | a NATURVETENSKAPx Biologix Biokemi och molekylärbiologi0 (SwePub)106022 hsv//swe |
| 650 | 7 | a NATURAL SCIENCESx Biological Sciencesx Biochemistry and Molecular Biology0 (SwePub)106022 hsv//eng |
| 653 | a eating disorders | |
| 653 | a genetic correlation | |
| 653 | a substance use | |
| 653 | a alcohol dependence | |
| 653 | a anorexia-nervosa | |
| 653 | a drug-use | |
| 653 | a environmental | |
| 653 | a contributions | |
| 653 | a bulimia-nervosa | |
| 653 | a women | |
| 653 | a gwas | |
| 653 | a twin | |
| 653 | a endocannabinoids | |
| 653 | a comorbidity | |
| 653 | a Biochemistry & Molecular Biology | |
| 653 | a Substance Abuse | |
| 700 | 1 | a Johnson, E. C.4 aut |
| 700 | 1 | a Chou, Y. L.4 aut |
| 700 | 1 | a Coleman, J. R. I.4 aut |
| 700 | 1 | a Landén, Mikael,d 1966u Gothenburg University,Göteborgs universitet,Karolinska Institutet,Institutionen för neurovetenskap och fysiologi, sektionen för psykiatri och neurokemi,Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry4 aut0 (Swepub:gu)xlandt |
| 700 | 1 | a Walters, R. K.4 aut |
| 700 | 1 | a Yilmaz, Z.4 aut |
| 700 | 1 | a Baker, J. H.4 aut |
| 700 | 1 | a Hubel, C.u Karolinska Institutet4 aut |
| 700 | 1 | a Gordon, S.4 aut |
| 700 | 1 | a Medland, S. E.4 aut |
| 700 | 1 | a Watson, H. J.4 aut |
| 700 | 1 | a Gaspar, H. A.4 aut |
| 700 | 1 | a Bryois, J.u Karolinska Institutet4 aut |
| 700 | 1 | a Adan, Roger A. H.u Gothenburg University,Göteborgs universitet,Institutionen för neurovetenskap och fysiologi,Institute of Neuroscience and Physiology4 aut0 (Swepub:gu)xadaro |
| 700 | 1 | a Leppa, V. M.u Karolinska Institutet4 aut |
| 700 | 1 | a Mattheisen, M.u Karolinska Institutet4 aut |
| 700 | 1 | a Ripke, S.4 aut |
| 700 | 1 | a Yao, S. Y.u Karolinska Institutet4 aut |
| 700 | 1 | a Giusti-Rodriguez, P.4 aut |
| 700 | 1 | a Hanscombe, K. B.4 aut |
| 700 | 1 | a Adan, Roger A. H.u Gothenburg University,Göteborgs universitet,Institutionen för neurovetenskap och fysiologi,Institute of Neuroscience and Physiology4 aut0 (Swepub:gu)xadaro |
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| 700 | 1 | a Landén, Mikael,d 1966u Gothenburg University,Göteborgs universitet,Institutionen för neurovetenskap och fysiologi, sektionen för psykiatri och neurokemi,Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry4 aut0 (Swepub:gu)xlandt |
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| 700 | 1 | a Martin, N. G.4 aut |
| 700 | 1 | a Sullivan, P. F.u Karolinska Institutet4 aut |
| 700 | 1 | a Kaprio, J.4 aut |
| 700 | 1 | a Breen, G.4 aut |
| 700 | 1 | a Gelernter, J.4 aut |
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| 710 | 2 | a Karolinska Institutetb Institutionen för neurovetenskap och fysiologi, sektionen för psykiatri och neurokemi4 org |
| 773 | 0 | t Addiction Biologyd : Wileyg 26:1q 26:1x 1355-6215x 1369-1600 |
| 856 | 4 | u https://escholarship.org/content/qt02g168jh/qt02g168jh.pdf?t=q6z0xq |
| 856 | 4 8 | u https://gup.ub.gu.se/publication/290860 |
| 856 | 4 8 | u https://doi.org/10.1111/adb.12880 |
| 856 | 4 8 | u http://kipublications.ki.se/Default.aspx?queryparsed=id:143007973 |
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- Courtet, P.
- Crow, S.
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