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Ligand-independent repression by the thyroid hormone receptor mediated by a nuclear receptor co-repressor

Hörlein, A. J. (author)
University of California, San Diego, USA
Näär, A. M. (author)
University of California, San Diego, USA
Heinzel, T. (author)
University of California, San Diego, USA
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Torchia, J. (author)
University of California, San Diego, USA
Gloss, B. (author)
University of California, San Diego, USA
Kurokawa, R. (author)
University of California, San Diego, USA
Ryan, A. (author)
University of California, San Diego, USA
Kamei, Y. (author)
University of California, San Diego, USA
Söderström, Mats (author)
University of California, San Diego, USA
Glass, C. K. (author)
University of California, San Diego, USA
Rosenfeld, M. G. (author)
University of California, San Diego, USA
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 (creator_code:org_t)
Nature Publishing Group, 1995
1995
English.
In: Nature. - : Nature Publishing Group. - 0028-0836 .- 1476-4687. ; 377:6548, s. 397-404
Related links:
https://urn.kb.se/re...
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https://doi.org/10.1...
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  • Journal article (peer-reviewed)
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  • Thyroid-hormone and retinoic-acid receptors exert their regulatory functions by acting as both activators and repressors of gene expression. A nuclear receptor co-repressor (N-CoR) of relative molecular mass 270K has been identified which mediates ligand-independent inhibition of gene transcription by these receptors, suggesting that the molecular mechanisms of repression by thyroid-hormone and retinoic-acid receptors are analogous to the co-repressor-dependent transcriptional inhibitory mechanisms of yeast and Drosophila.

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