WFRF:(Semnic Robert)
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| Fältnamn | Indikatorer | Metadata |
|---|---|---|
| 000 | 03904naa a2200421 4500 | |
| 001 | oai:DiVA.org:uu-439317 | |
| 003 | SwePub | |
| 008 | 210414s2020 | |||||||||||000 ||eng| | |
| 024 | 7 | a https://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-4393172 URI |
| 024 | 7 | a https://doi.org/10.1007/s00701-020-04425-32 DOI |
| 040 | a (SwePub)uu | |
| 041 | a engb eng | |
| 042 | 9 SwePub | |
| 072 | 7 | a ref2 swepub-contenttype |
| 072 | 7 | a art2 swepub-publicationtype |
| 100 | 1 | a Zetterling, Mariau Uppsala universitet,Enblad: Neurokirurgi4 aut0 (Swepub:uu)mazet677 |
| 245 | 1 0 | a Time course of neurological deficits after surgery for primary brain tumours |
| 264 | c 2020-07-02 | |
| 264 | 1 | b SPRINGER WIEN,c 2020 |
| 338 | a electronic2 rdacarrier | |
| 520 | a Background The postoperative course after surgery for primary brain tumours can be difficult to predict. We examined the time course of postoperative neurological deficits and analysed possible predisposing factors. Method Hundred adults with a radiological suspicion of low- or high-grade glioma were prospectively included and the postoperative course analysed. Possible predictors of postoperative neurological deterioration were evaluated. Results New postoperative neurologic deficits occurred in 37% of the patients, and in 4%, there were worsening of a preoperative deficit. In 78%, the deficits occurred directly after surgery. The probable cause of deterioration was EEG-verified seizures in 7, ischemic lesion in 5 and both in 1, resection of eloquent tissue in 6, resection close to eloquent tissue including SMA in 11 and postoperative haematoma in 1 patient. Seizures were the main cause of delayed neurological deterioration. Two-thirds of patients with postoperative deterioration showed complete regression of the deficits, and in 6% of all patients, there was a slight disturbance of the function after 3 months. Remaining deficits were found in 6% and only in patients with preoperative neurological deficits and high-grade tumours with mainly eloquent locations. Eloquent tumour location was a predictor of postoperative neurological deterioration and preoperative neurological deficits of remaining deficits. Conclusions Postoperative neurological deficits occurred in 41% and remained in 6% of patients. Remaining deficits were found in patients with preoperative neurological deficits and high-grade tumours with mainly eloquent locations. Eloquent tumour location was a predictor of neurological deterioration and preoperative neurological deficits of remaining deficits. | |
| 650 | 7 | a MEDICIN OCH HÄLSOVETENSKAPx Klinisk medicinx Kirurgi0 (SwePub)302122 hsv//swe |
| 650 | 7 | a MEDICAL AND HEALTH SCIENCESx Clinical Medicinex Surgery0 (SwePub)302122 hsv//eng |
| 653 | a Brain tumour surgery | |
| 653 | a Postoperative neurological deficit | |
| 653 | a Complications | |
| 653 | a Time course | |
| 700 | 1 | a Elf, Kristinu Uppsala universitet,Rostedt Punga: Klinisk neurofysiologi4 aut0 (Swepub:uu)krisjoha |
| 700 | 1 | a Semnic, Robertu Uppsala Univ, Dept Surg Sci, Radiol, S-75185 Uppsala, Sweden.4 aut |
| 700 | 1 | a Latini, Francesco,c M.D.d 1982-u Uppsala universitet,Enblad: Neurokirurgi4 aut0 (Swepub:uu)frala590 |
| 700 | 1 | a Ronne-Engström, Elisabethu Uppsala universitet,Enblad: Neurokirurgi4 aut0 (Swepub:uu)elisronn |
| 710 | 2 | a Uppsala universitetb Enblad: Neurokirurgi4 org |
| 773 | 0 | t Acta Neurochirurgicad : SPRINGER WIENg 162:12, s. 3005-3018q 162:12<3005-3018x 0001-6268x 0942-0940 |
| 856 | 4 | u https://doi.org/10.1007/s00701-020-04425-3y Fulltext |
| 856 | 4 | u https://uu.diva-portal.org/smash/get/diva2:1544114/FULLTEXT01.pdfx primaryx Raw objecty fulltext:print |
| 856 | 4 | u https://link.springer.com/content/pdf/10.1007/s00701-020-04425-3.pdf |
| 856 | 4 8 | u https://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-439317 |
| 856 | 4 8 | u https://doi.org/10.1007/s00701-020-04425-3 |
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