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FältnamnIndikatorerMetadata
00006400naa a2200613 4500
001oai:DiVA.org:uu-343327
003SwePub
008180312s2017 | |||||||||||000 ||eng|
0247 a https://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-3433272 URI
0247 a https://doi.org/10.1161/CIRCINTERVENTIONS.117.0055822 DOI
040 a (SwePub)uu
041 a engb eng
042 9 SwePub
072 7a ref2 swepub-contenttype
072 7a art2 swepub-publicationtype
1001 a Kolte, Dhavalu Brown Univ, Dept Med, Div Cardiol, Providence, RI 02912 USA.4 aut
2451 0a Culprit Vessel-Only Versus Multivessel Percutaneous Coronary Intervention in Patients With Cardiogenic Shock Complicating ST-Segment-Elevation Myocardial Infarction :b A Collaborative Meta-Analysis
264 1b LIPPINCOTT WILLIAMS & WILKINS,c 2017
338 a print2 rdacarrier
520 a Background The optimal revascularization strategy in patients with multivessel disease presenting with cardiogenic shock complicating ST-segment-elevation myocardial infarction remains unknown. Methods and Results Databases were searched from 1999 to October 2016. Studies comparing immediate/single-stage multivessel percutaneous coronary intervention (MV-PCI) versus culprit vessel-only PCI (CO-PCI) in patients with multivessel disease, ST-segment-elevation myocardial infarction, and cardiogenic shock were included. Primary end point was short-term (in-hospital or 30 days) mortality. Secondary end points included long-term mortality, cardiovascular death, reinfarction, and repeat revascularization. Safety end points were in-hospital stroke, renal failure, and major bleeding. The meta-analysis included 11 nonrandomized studies and 5850 patients (1157 MV-PCI and 4693 CO-PCI). There was no significant difference in short-term mortality with MV-PCI versus CO-PCI (odds ratio [OR], 1.08; 95% confidence interval [CI], 0.81-1.43; P=0.61). Similarly, there were no significant differences in long-term mortality (OR, 0.84; 95% CI, 0.54-1.30; P=0.43), cardiovascular death (OR, 0.72; 95% CI, 0.42-1.23; P=0.23), reinfarction (OR, 1.65; 95% CI, 0.84-3.26; P=0.15), or repeat revascularization (OR, 1.13; 95% CI, 0.76-1.69; P=0.54) between the 2 groups. There was a nonsignificant trend toward higher in-hospital stroke (OR, 1.64; 95% CI, 0.98-2.72; P=0.06) and renal failure (OR, 1.30; 95% CI, 0.98-1.72; P=0.06), with no difference in major bleeding (OR, 1.47; 95% CI, 0.39-5.63; P=0.57) with MV-PCI when compared with CO-PCI. Conclusions This meta-analysis of nonrandomized studies suggests that in patients with cardiogenic shock complicating ST-segment-elevation myocardial infarction, there may be no significant benefit with single-stage MV-PCI compared with CO-PCI. Given the limitations of observational data, randomized trials are needed to determine the role of MV-PCI in this setting.
650 7a MEDICIN OCH HÄLSOVETENSKAPx Klinisk medicinx Kardiologi0 (SwePub)302062 hsv//swe
650 7a MEDICAL AND HEALTH SCIENCESx Clinical Medicinex Cardiac and Cardiovascular Systems0 (SwePub)302062 hsv//eng
653 a cardiogenic shock
653 a complete revascularization
653 a mortality
653 a myocardial infarction
653 a percutaneous coronary intervention
653 a stroke
7001 a Sardar, Parthau Univ Utah, Dept Med, Div Cardiol, Salt Lake City, UT 84112 USA.4 aut
7001 a Khera, Sahilu New York Med Coll, Dept Med, Westchester Med Ctr, Div Cardiol, Valhalla, NY 10595 USA.4 aut
7001 a Zeymer, Uweu Inst Herzinfarktforsch Ludwigshafen, Dept Cardiol, Ludwigshafen, Germany.4 aut
7001 a Thiele, Holgeru Univ Hosp Schleswig Holstein, Univ Heart Ctr Lubeck, Med Clin 2, Dept Cardiol, Kiel, Germany.;German Cardiovasc Res Ctr DZHK, Partner Site Hamburg Kiel Lubeck, Hamburg, Germany.4 aut
7001 a Hochadel, Matthiasu Inst Herzinfarktforsch Ludwigshafen, Dept Cardiol, Ludwigshafen, Germany.4 aut
7001 a Radovanovic, Draganau Univ Zurich, AMIS Plus Data Ctr, Zurich, Switzerland.4 aut
7001 a Erne, Paulu Univ Zurich, AMIS Plus Data Ctr, Zurich, Switzerland.4 aut
7001 a Hambraeus, Kristina,d 1970-u Uppsala universitet,Centrum för klinisk forskning Dalarna,Falun Cent Hosp, Dept Cardiol, Falun, Sweden.4 aut0 (Swepub:uu)kriha420
7001 a James, Stefan K,d 1964-u Uppsala universitet,Kardiologi4 aut0 (Swepub:uu)stjam367
7001 a Claessen, Bimmer E.u Univ Amsterdam, Acad Med Ctr, Dept Cardiol, Amsterdam, Netherlands.4 aut
7001 a Henriques, Jose P. S.u Univ Amsterdam, Acad Med Ctr, Dept Cardiol, Amsterdam, Netherlands.4 aut
7001 a Mylotte, Darrenu Natl Univ Ireland, SAOLTA Healthcare Grp, Galway Univ Hosp, Dept Cardiol, Galway, Ireland.4 aut
7001 a Garot, Philippeu Hop Prive Jacques Cartier, Inst Cardiovasc Paris Sud, Ramsay Gen Sante, Dept Cardiol, Massy, France.4 aut
7001 a Aronow, Wilbert S.u New York Med Coll, Dept Med, Westchester Med Ctr, Div Cardiol, Valhalla, NY 10595 USA.4 aut
7001 a Owan, Theophilusu Univ Utah, Dept Med, Div Cardiol, Salt Lake City, UT 84112 USA.4 aut
7001 a Jain, Diwakaru New York Med Coll, Dept Med, Westchester Med Ctr, Div Cardiol, Valhalla, NY 10595 USA.4 aut
7001 a Panza, Julio A.u New York Med Coll, Dept Med, Westchester Med Ctr, Div Cardiol, Valhalla, NY 10595 USA.4 aut
7001 a Frishman, William H.u New York Med Coll, Dept Med, Westchester Med Ctr, Div Cardiol, Valhalla, NY 10595 USA.4 aut
7001 a Fonarow, Gregg C.u Univ Calif Los Angeles, David Geffen Sch Med, Dept Med, Div Cardiol, Los Angeles, CA 90095 USA.4 aut
7001 a Bhatt, Deepak L.u Harvard Med Sch, Brigham & Womens Hosp, Heart & Vasc Ctr, Dept Med,Div Cardiol, Boston, MA USA.4 aut
7001 a Aronow, Herbert D.u Brown Univ, Dept Med, Div Cardiol, Providence, RI 02912 USA.4 aut
7001 a Abbott, J. Dawnu Brown Univ, Dept Med, Div Cardiol, Providence, RI 02912 USA.4 aut
7102 a Brown Univ, Dept Med, Div Cardiol, Providence, RI 02912 USA.b Univ Utah, Dept Med, Div Cardiol, Salt Lake City, UT 84112 USA.4 org
7730 t Circulation. Cardiovascular Interventionsd : LIPPINCOTT WILLIAMS & WILKINSg 10:11q 10:11x 1941-7640x 1941-7632
8564 8u https://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-343327
8564 8u https://doi.org/10.1161/CIRCINTERVENTIONS.117.005582

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