Hypovolaemic stress induced glycogenolysis, isolated liver perfusion study

↓ Direkt till sidans innehåll
↓ Direkt till sidans sekundära innehåll (sidomenyn)

Sökning: WFRF:(Nylander Per) > Hypovolaemic stress...

LIBRIS Formathandbok (Information om MARC21)

Fältnamn	Indikatorer	Metadata
000		03049naa a2200337 4500
001		oai:DiVA.org:oru-63831
003		SwePub
008		180104s1987 \| \|\|\|\|\|\|\|\|\|\|\|000 \|\|eng\|
024	7	a https://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-638312 URI
024	7	a https://doi.org/10.1007/BF018556562 DOI
040		a (SwePub)oru
041		a engb eng
042		9 SwePub
072	7	a ref2 swepub-contenttype
072	7	a art2 swepub-publicationtype
100	1	a Boija, Per Olovu Department. of Surgery, Karolinska Hospital, Stockholm, Sweden4 aut
245	1 0	a Hypovolaemic stress induced glycogenolysis, isolated liver perfusion study
264	1	b Springer,c 1987
338		a print2 rdacarrier
520		a Intrinsic hepatic glycogenolysis was examined after hypovolemic stress. Hemorrhagic hypotension of 70 (P70) and 40 mm Hg (P40) for 60 min was inflicted for two postprandial groups and of 70 mm Hg (S70) in a 24-h starved group. The results were compared with three control groups; one postprandial (Pc), one 24-h starved (Sc), and one starved for 9 h (Sc: 9) to mimic the glycogen depletion produced by 70 mm Hg hemorrhagic hypotension. Glucose output was studied in vitro using av recirculating isolated liver perfusion system with a perfusate free of glucose and endocrine stimulation. Liver glycogen determination was made before perfusion start. Although the glycogen stores were decreased after hemorrhage glucose yield was increased (P70) and unchanged (P40) as compared to controls (Pc and Sc: 9). Both starved groups delivered small amounts of glucose, but the released fraction of the S70 group was more than twice that from the Sc group. These data suggest a liver enzyme activation with increased velocity of the enzymesubstrate reactions responsible for glycogen degradation, induced during in vivo hemorrhage and persisting for at least 30 min in vitro perfusion.
650	7	a MEDICIN OCH HÄLSOVETENSKAPx Medicinska och farmaceutiska grundvetenskaperx Fysiologi0 (SwePub)301062 hsv//swe
650	7	a MEDICAL AND HEALTH SCIENCESx Basic Medicinex Physiology0 (SwePub)301062 hsv//eng
650	7	a MEDICIN OCH HÄLSOVETENSKAPx Klinisk medicinx Kirurgi0 (SwePub)302122 hsv//swe
650	7	a MEDICAL AND HEALTH SCIENCESx Clinical Medicinex Surgery0 (SwePub)302122 hsv//eng
700	1	a Ljungqvist, Olle,d 1954-u Örebro universitet,Institutionen för medicinska vetenskaper,Department. of Surgery, Karolinska Hospital, Stockholm, Sweden4 aut0 (Swepub:oru)olt
700	1	a Nylander, Gunnaru Department. of Surgery, Karolinska Hospital, Stockholm, Sweden4 aut
700	1	a Ware, Jamesu Department. of Surgery, Karolinska Hospital, Stockholm, Sweden4 aut
710	2	a Department. of Surgery, Karolinska Hospital, Stockholm, Swedenb Institutionen för medicinska vetenskaper4 org
773	0	t Research in experimental medicined : Springerg 187:5, s. 315-322q 187:5<315-322x 0300-9130x 1433-8580
856	4 8	u https://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-63831
856	4 8	u https://doi.org/10.1007/BF01855656

Hitta via bibliotek

Research in experimental medicine (Sök värdpublikationen i LIBRIS)

Till lärosätets databas

Hitta mer i SwePub

Av författaren/redakt...: Boija, Per Olov; Ljungqvist, Olle ...; Nylander, Gunnar; Ware, James

Om ämnet

MEDICIN OCH HÄLSOVETENSKAP: MEDICIN OCH HÄLS ...; och Medicinska och f ...; och Fysiologi

MEDICIN OCH HÄLSOVETENSKAP: MEDICIN OCH HÄLS ...; och Klinisk medicin; och Kirurgi

Artiklar i publikationen: Research in expe ...

Av lärosätet: Örebro universitet

Sök utanför SwePub

Sök vidare i:: Google; Google Book Search; Google Scholar

Kungliga biblioteket hanterar dina personuppgifter i enlighet med EU:s dataskyddsförordning (2018), GDPR. Läs mer om hur det funkar här.
Så här hanterar KB dina uppgifter vid användning av denna tjänst.

LIBRIS.kb.se