Search: WFRF:(Janssen A.) > (2020-2024) > Virtual Ontogeny of...
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000 | 10598naa a2202713 4500 | |
001 | oai:gup.ub.gu.se/320906 | |
003 | SwePub | |
008 | 240910s2022 | |||||||||||000 ||eng| | |
009 | oai:prod.swepub.kib.ki.se:151236548 | |
024 | 7 | a https://gup.ub.gu.se/publication/3209062 URI |
024 | 7 | a https://doi.org/10.1016/j.biopsych.2022.02.9592 DOI |
024 | 7 | a http://kipublications.ki.se/Default.aspx?queryparsed=id:1512365482 URI |
040 | a (SwePub)gud (SwePub)ki | |
041 | a eng | |
042 | 9 SwePub | |
072 | 7 | a ref2 swepub-contenttype |
072 | 7 | a art2 swepub-publicationtype |
100 | 1 | a Patel, Y.4 aut |
245 | 1 0 | a Virtual Ontogeny of Cortical Growth Preceding Mental Illness |
264 | 1 | b Elsevier BV,c 2022 |
520 | a Background: Morphology of the human cerebral cortex differs across psychiatric disorders, with neurobiology and developmental origins mostly undetermined. Deviations in the tangential growth of the cerebral cortex during pre/perinatal periods may be reflected in individual variations in cortical surface area later in life. Methods: Interregional profiles of group differences in surface area between cases and controls were generated using T1-weighted magnetic resonance imaging from 27,359 individuals including those with attention-deficit/hyperactivity disorder, autism spectrum disorder, bipolar disorder, major depressive disorder, schizophrenia, and high general psychopathology (through the Child Behavior Checklist). Similarity of interregional profiles of group differences in surface area and prenatal cell-specific gene expression was assessed. Results: Across the 11 cortical regions, group differences in cortical area for attention-deficit/hyperactivity disorder, schizophrenia, and Child Behavior Checklist were dominant in multimodal association cortices. The same interregional profiles were also associated with interregional profiles of (prenatal) gene expression specific to proliferative cells, namely radial glia and intermediate progenitor cells (greater expression, larger difference), as well as differentiated cells, namely excitatory neurons and endothelial and mural cells (greater expression, smaller difference). Finally, these cell types were implicated in known pre/perinatal risk factors for psychosis. Genes coexpressed with radial glia were enriched with genes implicated in congenital abnormalities, birth weight, hypoxia, and starvation. Genes coexpressed with endothelial and mural genes were enriched with genes associated with maternal hypertension and preterm birth. Conclusions: Our findings support a neurodevelopmental model of vulnerability to mental illness whereby prenatal risk factors acting through cell-specific processes lead to deviations from typical brain development during pregnancy. | |
650 | 7 | a MEDICIN OCH HÄLSOVETENSKAPx Klinisk medicinx Psykiatri0 (SwePub)302152 hsv//swe |
650 | 7 | a MEDICAL AND HEALTH SCIENCESx Clinical Medicinex Psychiatry0 (SwePub)302152 hsv//eng |
653 | a Cortical growth | |
653 | a Cortical surface area | |
653 | a Mental illness | |
653 | a Neurodevelopment | |
653 | a Neurogenesis | |
653 | a Psychiatric disorders | |
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700 | 1 | a Landén, Mikael,d 1966u Karolinska Institutet,Gothenburg University,Göteborgs universitet,Institutionen för neurovetenskap och fysiologi, sektionen för psykiatri och neurokemi,Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry4 aut0 (Swepub:gu)xlandt |
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710 | 2 | a Karolinska Institutetb Institutionen för neurovetenskap och fysiologi, sektionen för psykiatri och neurokemi4 org |
773 | 0 | t Biological Psychiatryd : Elsevier BVg 92:4, s. 299-313q 92:4<299-313x 0006-3223x 1873-2402 |
856 | 4 8 | u https://gup.ub.gu.se/publication/320906 |
856 | 4 8 | u https://doi.org/10.1016/j.biopsych.2022.02.959 |
856 | 4 8 | u http://kipublications.ki.se/Default.aspx?queryparsed=id:151236548 |
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