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Interleukin-17A Promotes Neutrophilia in Acute Exacerbation of Chronic Obstructive Pulmonary Disease.

Roos, Abraham (author)
Lund University,Lunds universitet,Luftvägsinflammation,Forskargrupper vid Lunds universitet,Airway Inflammation and Immunology,Lund University Research Groups,McMaster University
Sethi, Sanjay (author)
Nikota, Jake (author)
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Wrona, Catherine T (author)
Dorrington, Michael G (author)
Sandén, Caroline (author)
Lund University,Lunds universitet,Luftvägsinflammation,Forskargrupper vid Lunds universitet,Airway Inflammation and Immunology,Lund University Research Groups
Bauer, Carla Mt (author)
Shen, Pamela (author)
Bowdish, Dawn (author)
Stevenson, Christopher S (author)
Erjefält, Jonas (author)
Lund University,Lunds universitet,Luftvägsinflammation,Forskargrupper vid Lunds universitet,Airway Inflammation and Immunology,Lund University Research Groups
Stampfli, Martin R (author)
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 (creator_code:org_t)
2015
2015
English.
In: American Journal of Respiratory and Critical Care Medicine. - 1535-4970. ; 192:4, s. 428-437
  • Journal article (peer-reviewed)
Abstract Subject headings
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  • Nontypeable Haemophilus influenzae (NTHi) causes acute exacerbation of chronic obstructive pulmonary disease (AECOPD). Interleukin (IL)-17A is central for neutrophilic inflammation, and has been linked to COPD pathogenesis.

Subject headings

MEDICIN OCH HÄLSOVETENSKAP  -- Medicinska och farmaceutiska grundvetenskaper -- Immunologi inom det medicinska området (hsv//swe)
MEDICAL AND HEALTH SCIENCES  -- Basic Medicine -- Immunology in the medical area (hsv//eng)

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