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Search: WFRF:(Boutron Ruault M C.) > (2010-2014) > Tea and coffee cons...

LIBRIS Formathandbok  (Information om MARC21)
FältnamnIndikatorerMetadata
00006513naa a2201213 4500
001oai:gup.ub.gu.se/201530
003SwePub
008240528s2014 | |||||||||||000 ||eng|
009oai:lup.lub.lu.se:0e6b1c83-3765-4075-b175-e8e1a6482afb
009oai:DiVA.org:umu-86431
009oai:prod.swepub.kib.ki.se:129423036
0247 a https://gup.ub.gu.se/publication/2015302 URI
0247 a https://doi.org/10.1002/ijc.287892 DOI
0247 a https://lup.lub.lu.se/record/45917532 URI
0247 a https://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-864312 URI
0247 a http://kipublications.ki.se/Default.aspx?queryparsed=id:1294230362 URI
040 a (SwePub)gud (SwePub)lud (SwePub)umud (SwePub)ki
041 a eng
042 9 SwePub
072 7a ref2 swepub-contenttype
072 7a art2 swepub-publicationtype
1001 a Zamora-Ros, R.4 aut
2451 0a Tea and coffee consumption and risk of esophageal cancer: The European prospective investigation into cancer and nutrition study
264 c 2014-02-28
264 1b Wiley,c 2014
520 a Epidemiological data regarding tea and coffee consumption and risk of esophageal cancer (EC) is still inconclusive. We examined the association of tea and coffee consumption with EC risk among 442,143 men and women without cancer at baseline from 9 countries of the European Prospective Investigation into Cancer and Nutrition. Tea and coffee intakes were recorded using country-specific validated dietary questionnaires. Cox regression models were used to analyze the relationships between tea and coffee intake and EC risk. During a mean follow-up of 11.1 years, 339 participants developed EC, of which 142 were esophageal adenocarcinoma (EAC) and 174 were esophageal squamous cell carcinoma (ESCC). In the multivariable models, no significant associations between tea (mostly black tea), and coffee intake and risk of EC, EAC and ESCC were observed. In stratified analyses, among men coffee consumption was inversely related to ESCC (HR for comparison of extreme tertiles 0.42, 95% CI 0.20-0.88; p-trend = 0.022), but not among women. In current smokers, a significant and inverse association was observed between ESCC risk and tea (HR 0.46, 95% CI 0.23-0.93; p-trend = 0.053) and coffee consumption (HR 0.37, 95% CI 0.19-0.73; p-trend = 0.011). However, no statistically significant findings were observed using the continuous variable (per 100 mL/d). These data did not show a significant association between tea and coffee consumption and EC, EAC and ESCC, although a decreased risk of ESCC among men and current smokers is suggested, but need to be confirmed in further prospective studies including more cases.
650 7a MEDICIN OCH HÄLSOVETENSKAPx Klinisk medicinx Cancer och onkologi0 (SwePub)302032 hsv//swe
650 7a MEDICAL AND HEALTH SCIENCESx Clinical Medicinex Cancer and Oncology0 (SwePub)302032 hsv//eng
650 7a MEDICIN OCH HÄLSOVETENSKAPx Hälsovetenskapx Folkhälsovetenskap, global hälsa, socialmedicin och epidemiologi0 (SwePub)303022 hsv//swe
650 7a MEDICAL AND HEALTH SCIENCESx Health Sciencesx Public Health, Global Health, Social Medicine and Epidemiology0 (SwePub)303022 hsv//eng
650 7a MEDICIN OCH HÄLSOVETENSKAPx Hälsovetenskapx Näringslära0 (SwePub)303042 hsv//swe
650 7a MEDICAL AND HEALTH SCIENCESx Health Sciencesx Nutrition and Dietetics0 (SwePub)303042 hsv//eng
653 a tea
653 a coffee
653 a esophageal cancer
653 a smoking status
653 a EPIC
653 a GREEN TEA
653 a ADENOCARCINOMA RISK
653 a ALCOHOL-DRINKING
653 a GASTRIC CARDIA
653 a TRACT
653 a CANCER
653 a POLYPHENOLS
653 a METAANALYSIS
653 a COHORT
653 a CHINA
653 a MEN
653 a Oncology
653 a tea
653 a näringslära
653 a Public health
7001 a Lujan-Barroso, L.4 aut
7001 a Bueno-de-Mesquita, H. B.4 aut
7001 a Dik, V. K.4 aut
7001 a Boeing, H.4 aut
7001 a Steffen, A.4 aut
7001 a Tjonneland, A.4 aut
7001 a Olsen, A.4 aut
7001 a Bech, B. H.4 aut
7001 a Overvad, K.4 aut
7001 a Boutron-Ruault, M. C.4 aut
7001 a Racine, A.4 aut
7001 a Fagherazzi, G.4 aut
7001 a Kuhn, T.4 aut
7001 a Katzke, V.4 aut
7001 a Trichopoulou, A.4 aut
7001 a Lagiou, P.4 aut
7001 a Trichopoulos, D.4 aut
7001 a Tumino, R.4 aut
7001 a Panico, S.4 aut
7001 a Vineis, P.4 aut
7001 a Grioni, S.4 aut
7001 a Palli, D.4 aut
7001 a Weiderpass, E.u Karolinska Institutet4 aut
7001 a Skeie, G.4 aut
7001 a Huerta, J. M.4 aut
7001 a Sanchez, M. J.4 aut
7001 a Arguelles, M.4 aut
7001 a Amiano, P.4 aut
7001 a Ardanaz, E.4 aut
7001 a Nilsson, Lena Maria,d 1965-u Umeå universitet,Näringsforskning,Arktiskt centrum vid Umeå universitet (Arcum),nutrition4 aut0 (Swepub:umu)leni0002
7001 a Wallner, Bengt,d 1962-u Umeå universitet,Kirurgi4 aut0 (Swepub:umu)bewa0005
7001 a Lindkvist, Björnu Gothenburg University,Göteborgs universitet,Institutionen för medicin,Institute of Medicine4 aut0 (Swepub:gu)xlibjo
7001 a Wallström, Peteru Lund University,Lunds universitet,Nutritionsepidemiologi,Forskargrupper vid Lunds universitet,Nutrition Epidemiology,Lund University Research Groups4 aut0 (Swepub:lu)pe6853wa
7001 a Peeters, P. H. M.4 aut
7001 a Key, T. J.4 aut
7001 a Khaw, K. T.4 aut
7001 a Wareham, N. J.4 aut
7001 a Freisling, H.4 aut
7001 a Stepien, M.4 aut
7001 a Ferrari, P.4 aut
7001 a Gunter, M. J.4 aut
7001 a Murphy, N.4 aut
7001 a Riboli, E.4 aut
7001 a Gonzalez, C. A.4 aut
7102 a Karolinska Institutetb Näringsforskning4 org
7730 t International Journal of Cancerd : Wileyg 135:6, s. 1470-1479q 135:6<1470-1479x 0020-7136x 1097-0215
8564 u http://dx.doi.org/10.1002/ijc.28789y FULLTEXT
8564 8u https://gup.ub.gu.se/publication/201530
8564 8u https://doi.org/10.1002/ijc.28789
8564 8u https://lup.lub.lu.se/record/4591753
8564 8u https://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-86431
8564 8u http://kipublications.ki.se/Default.aspx?queryparsed=id:129423036

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