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Sökning: WFRF:(Courtney M. J.) > (2015-2019) > Interaction between...

LIBRIS Formathandbok  (Information om MARC21)
FältnamnIndikatorerMetadata
00008314naa a2201009 4500
001oai:DiVA.org:umu-138555
003SwePub
008170914s2017 | |||||||||||000 ||eng|
009oai:lup.lub.lu.se:755e4996-b952-444f-93a5-777faa86bc80
0247 a https://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1385552 URI
0247 a https://doi.org/10.3945/ajcn.116.1500942 DOI
0247 a https://lup.lub.lu.se/record/755e4996-b952-444f-93a5-777faa86bc802 URI
040 a (SwePub)umud (SwePub)lu
041 a engb eng
042 9 SwePub
072 7a ref2 swepub-contenttype
072 7a for2 swepub-publicationtype
1001 a Li, Sherly X.u University of Cambridge4 aut
2451 0a Interaction between genes and macronutrient intake on the risk of developing type 2 diabetes :b systematic review and findings from European Prospective Investigation into Cancer (EPIC)-InterAct
264 1b American society for nutrition,c 2017
338 a print2 rdacarrier
520 a Background: Gene-diet interactions have been reported to contribute to the development of type 2 diabetes (T2D). However, to our knowledge, few examples have been consistently replicated to date. Objective: We aimed to identify existing evidence for genemacronutrient interactions and T2D and to examine the reported interactions in a large-scale study. Design: We systematically reviewed studies reporting genemacronutrient interactions and T2D. We searched the MEDLINE, Human Genome Epidemiology Network, and WHO International Clinical Trials Registry Platform electronic databases to identify studies published up to October 2015. Eligibility criteria included assessment of macronutrient quantity (e.g., total carbohydrate) or indicators of quality (e. g., dietary fiber) by use of self-report or objective biomarkers of intake. Interactions identified in the review were subsequently examined in the EPIC (European Prospective Investigation into Cancer)-InterAct case-cohort study (n = 21,148, with 9403 T2D cases; 8 European countries). Prentice-weighted Cox regression was used to estimate countryspecific HRs, 95% CIs, and P-interaction values, which were then pooled by random-effects meta-analysis. A primary model was fitted by using the same covariates as reported in the published studies, and a second model adjusted for additional covariates and estimated the effects of isocaloric macronutrient substitution. Results: Thirteen observational studies met the eligibility criteria (n < 1700 cases). Eight unique interactions were reported to be significant between macronutrients [carbohydrate, fat, saturated fat, dietary fiber, and glycemic load derived from self-report of dietary intake and circulating n-3 (v-3) polyunsaturated fatty acids] and genetic variants in or near transcription factor 7-like 2 (TCF7L2), gastric inhibitory polypeptide receptor (GIPR), caveolin 2 (CAV2), and peptidase D (PEPD) (P-interaction, 0.05). We found no evidence of interaction when we tried to replicate previously reported interactions. In addition, no interactions were detected in models with additional covariates. Conclusions: Eight gene-macronutrient interactions were identified for the risk of T2D from the literature. These interactions were not replicated in the EPIC-InterAct study, which mirrored the analyses undertaken in the original reports. Our findings highlight the importance of independent replication of reported interactions.
650 7a MEDICIN OCH HÄLSOVETENSKAPx Hälsovetenskapx Näringslära0 (SwePub)303042 hsv//swe
650 7a MEDICAL AND HEALTH SCIENCESx Health Sciencesx Nutrition and Dietetics0 (SwePub)303042 hsv//eng
650 7a MEDICIN OCH HÄLSOVETENSKAPx Medicinska och farmaceutiska grundvetenskaperx Medicinsk genetik0 (SwePub)301072 hsv//swe
650 7a MEDICAL AND HEALTH SCIENCESx Basic Medicinex Medical Genetics0 (SwePub)301072 hsv//eng
653 a macronutrient
653 a diet
653 a gene
653 a diabetes
653 a interaction
653 a effect modification
653 a systematic review
653 a replication
653 a Diabetes
653 a Diet
653 a Effect modification
653 a Gene
653 a Interaction
653 a Macronutrient
653 a Replication
653 a Systematic review
7001 a Imamura, Fumiakiu University of Cambridge4 aut
7001 a Ye, Zhengu University of Cambridge4 aut
7001 a Schulze, Matthias B.u German Center for Diabetes Research,German Institute of Human Nutrition4 aut
7001 a Zheng, Jushengu University of Cambridge4 aut
7001 a Ardanaz, Evau Health Research Institute of Navarra (IDISNA)4 aut
7001 a Arriola, Larraitzu Public Health Division of Gipuzkoa4 aut
7001 a Boeing, Heineru German Institute of Human Nutrition4 aut
7001 a Dow, Courtneyu University of Paris-Saclay,Centre for Research in Epidemiology and Population Health (CESP)4 aut
7001 a Fagherazzi, Guyu University of Paris-Saclay,Centre for Research in Epidemiology and Population Health (CESP)4 aut
7001 a Franks, Paul W.u Lund University,Lunds universitet,Umeå universitet,Medicin,Lund Univ, Sweden,Genetisk och molekylär epidemiologi,Forskargrupper vid Lunds universitet,Genetic and Molecular Epidemiology,Lund University Research Groups4 aut0 (Swepub:lu)med-plf
7001 a Agudo, Antoniou Catalan Institute of Oncology,German Cancer Research Centre,University of Cambridge4 aut
7001 a Grioni, Sara4 aut
7001 a Kaaks, Rudolfu German Cancer Research Centre4 aut
7001 a Katzke, Verena A.4 aut
7001 a Key, Timothy J.u University of Oxford4 aut
7001 a Khaw, Kay Teeu University of Cambridge4 aut
7001 a Mancini, Francesca R.u Centre for Research in Epidemiology and Population Health (CESP),University of Paris-Saclay4 aut
7001 a Navarro, Carmenu University of Murcia,Murcia Regional Health Council4 aut
7001 a Nilsson, Peter M.u Lund University,Lunds universitet,Internmedicin - epidemiologi,Forskargrupper vid Lunds universitet,Internal Medicine - Epidemiology,Lund University Research Groups4 aut0 (Swepub:lu)medf-pni
7001 a Onland-Moret, N. Charlotteu University Medical Center Utrecht4 aut
7001 a Overvad, Kimu Aalborg University,Aarhus University4 aut
7001 a Palli, Domenicou Cancer Research and Prevention Institute (ISPO)4 aut
7001 a Panico, Salvatoreu University of Naples Federico II4 aut
7001 a Quiros, J. Ramon4 aut
7001 a Rolandsson, Olovu Umeå University,Umeå universitet,Allmänmedicin4 aut0 (Swepub:umu)olro0005
7001 a Sacerdote, Carlottau Centre for Cancer Epidemiology and Prevention (CPO Piemonte),Citta' della Salute e della Scienza Hospital-University of Turin,Human Genetics Foundation (HuGeF)4 aut
7001 a Sanchez, Maria-Joseu Andalusian School Of Public Health (EASP)4 aut
7001 a Slimani, Nadiau International Agency for Research on Cancer, World Health Organization4 aut
7001 a Sluijs, Ivonneu University Medical Center Utrecht4 aut
7001 a Spijkerman, Annemieke M. W.u National Institute for Public Health and the Environment (RIVM)4 aut
7001 a Tjonneland, Anneu Danish Cancer Society4 aut
7001 a Tumino, Rosario4 aut
7001 a Sharp, Stephen J.u University of Cambridge4 aut
7001 a Riboli, Eliou Imperial College London4 aut
7001 a Langenberg, Claudiau University of Cambridge4 aut
7001 a Scott, Robert A.4 aut
7001 a Forouhi, Nita G.u University of Cambridge4 aut
7001 a Wareham, Nicholas J.u University of Cambridge4 aut
7102 a University of Cambridgeb German Center for Diabetes Research4 org
7730 t American Journal of Clinical Nutritiond : American society for nutritiong 106:1, s. 263-275q 106:1<263-275x 0002-9165x 1938-3207
8564 u https://academic.oup.com/ajcn/article-pdf/106/1/263/23802601/ajcn150094.pdf
8564 u http://dx.doi.org/10.3945/ajcn.116.150094x freey FULLTEXT
8564 8u https://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-138555
8564 8u https://doi.org/10.3945/ajcn.116.150094
8564 8u https://lup.lub.lu.se/record/755e4996-b952-444f-93a5-777faa86bc80

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