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LIBRIS Formathandbok  (Information om MARC21)
FältnamnIndikatorerMetadata
00005097naa a2200829 4500
001oai:lup.lub.lu.se:6784bb6f-7516-4b0a-a1bf-7eff4bad9cf0
003SwePub
008160401s2015 | |||||||||||000 ||eng|
009oai:prod.swepub.kib.ki.se:131613010
0247 a https://lup.lub.lu.se/record/77797342 URI
0247 a https://doi.org/10.1136/annrheumdis-2013-2039332 DOI
0247 a http://kipublications.ki.se/Default.aspx?queryparsed=id:1316130102 URI
040 a (SwePub)lud (SwePub)ki
041 a engb eng
042 9 SwePub
072 7a art2 swepub-publicationtype
072 7a ref2 swepub-contenttype
1001 a Parker, Ben4 aut
2451 0a Impact of early disease factors on metabolic syndrome in systemic lupus erythematosus: data from an international inception cohort
264 c 2014-04-01
264 1b BMJ,c 2015
338 a electronic2 rdacarrier
520 a Background The metabolic syndrome (MetS) may contribute to the increased cardiovascular risk in systemic lupus erythematosus (SLE). We examined the association between MetS and disease activity, disease phenotype and corticosteroid exposure over time in patients with SLE. Methods Recently diagnosed (< 15 months) patients with SLE from 30 centres across 11 countries were enrolled into the Systemic Lupus International Collaborating Clinics (SLICC) Inception Cohort from 2000 onwards. Baseline and annual assessments recorded clinical, laboratory and therapeutic data. A longitudinal analysis of factors associated with MetS in the first 2 years of follow-up was performed using random effects logistic regression. Results We studied 1150 patients with a mean (SD) age of 34.9 (13.6) years and disease duration at enrolment of 24.2 (18.0) weeks. In those with complete data, MetS prevalence was 38.2% at enrolment, 34.8% at year 1 and 35.4% at year 2. In a multivariable random effects model that included data from all visits, prior MetS status, baseline renal disease, SLICC Damage Index > 1, higher disease activity, increasing age and Hispanic or Black African race/ethnicity were independently associated with MetS over the first 2 years of follow-up in the cohort. Conclusions MetS is a persistent phenotype in a significant proportion of patients with SLE. Renal lupus, active inflammatory disease and damage are SLE-related factors that drive MetS development while antimalarial agents appear to be protective from early in the disease course.
650 7a MEDICIN OCH HÄLSOVETENSKAPx Klinisk medicinx Reumatologi och inflammation0 (SwePub)302102 hsv//swe
650 7a MEDICAL AND HEALTH SCIENCESx Clinical Medicinex Rheumatology and Autoimmunity0 (SwePub)302102 hsv//eng
7001 a Urowitz, Murray B.4 aut
7001 a Gladman, Dafna D.4 aut
7001 a Lunt, Mark4 aut
7001 a Donn, Rachelle4 aut
7001 a Bae, Sang-Cheol4 aut
7001 a Sanchez-Guerrero, Jorge4 aut
7001 a Romero-Diaz, Juanita4 aut
7001 a Gordon, Caroline4 aut
7001 a Wallace, Daniel J.4 aut
7001 a Clarke, Ann E.4 aut
7001 a Bernatsky, Sasha4 aut
7001 a Ginzler, Ellen M.4 aut
7001 a Isenberg, David A.4 aut
7001 a Rahman, Anisur4 aut
7001 a Merrill, Joan T.4 aut
7001 a Alarcon, Graciela S.4 aut
7001 a Fessler, Barri J.4 aut
7001 a Fortin, Paul R.4 aut
7001 a Hanly, John G.4 aut
7001 a Petri, Michelle4 aut
7001 a Steinsson, Kristjan4 aut
7001 a Dooley, Mary Anne4 aut
7001 a Manzi, Susan4 aut
7001 a Khamashta, Munther A.4 aut
7001 a Ramsey-Goldman, Rosalind4 aut
7001 a Zoma, Asad A.4 aut
7001 a Sturfelt, Gunnaru Lund University,Lunds universitet,Reumatologi och molekylär skelettbiologi,Sektion III,Institutionen för kliniska vetenskaper, Lund,Medicinska fakulteten,Rheumatology,Section III,Department of Clinical Sciences, Lund,Faculty of Medicine4 aut0 (Swepub:lu)reum-gst
7001 a Nived, Ola4 aut
7001 a Aranow, Cynthia4 aut
7001 a Mackay, Meggan4 aut
7001 a Ramos-Casals, Manuel4 aut
7001 a van Vollenhoven, Ronald F.u Karolinska Institutet4 aut
7001 a Kalunian, Kenneth C.4 aut
7001 a Ruiz-Irastorza, Guillermo4 aut
7001 a Lim, S. Sam4 aut
7001 a Kamen, Diane L.4 aut
7001 a Peschken, Christine A.4 aut
7001 a Inanc, Murat4 aut
7001 a Bruce, Ian N.4 aut
7102 a Reumatologi och molekylär skelettbiologib Sektion III4 org
7730 t Annals of the Rheumatic Diseasesd : BMJg 74:8, s. 1530-1536q 74:8<1530-1536x 1468-2060x 0003-4967
8564 u https://portal.research.lu.se/files/3018712/8626211x primaryx freey FULLTEXT
8564 u http://dx.doi.org/10.1136/annrheumdis-2013-203933x freey FULLTEXT
8564 u https://ard.bmj.com/content/74/8/1530.full.pdf
8564 8u https://lup.lub.lu.se/record/7779734
8564 8u https://doi.org/10.1136/annrheumdis-2013-203933
8564 8u http://kipublications.ki.se/Default.aspx?queryparsed=id:131613010

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