Pharmacogenomics and Personalized Medicine in Mood Disorders

↓ Direkt till sidans innehåll
↓ Direkt till sidans sekundära innehåll (sidomenyn)

Sökning: WFRF:(Archer S.) > Pharmacogenomics an...

LIBRIS Formathandbok (Information om MARC21)

Fältnamn	Indikatorer	Metadata
000		02793naa a2200289 4500
001		oai:gup.ub.gu.se/189037
003		SwePub
008		240528s2013 \| \|\|\|\|\|\|\|\|\|\|\|000 \|\|eng\|
024	7	a https://gup.ub.gu.se/publication/1890372 URI
040		a (SwePub)gu
041		a eng
042		9 SwePub
072	7	a ref2 swepub-contenttype
072	7	a kap2 swepub-publicationtype
100	1	a Archer, Trevor,d 1949u Gothenburg University,Göteborgs universitet,Psykologiska institutionen,Department of Psychology4 aut0 (Swepub:gu)xarctr
245	1 0	a Pharmacogenomics and Personalized Medicine in Mood Disorders
264	1	b Springer,c 2013
520		a Neurotoxic vulnerability that putatively contributes to the etiopathogenesis of schizophrenia spectrum disorders encompasses perinatal adversity, genetic linkage, epigenetic disadvantage, and neurodegenerative propensities that affect both symptom domains, positive, negative, and cognitive and biomarkers of the disorder. Molecular and cellular apoptosis/excitotoxicity that culminates in regional brain loss, reductions reelin expression, trophic disruption, perinatal adversity, glycogen kinase-3 dysregulation, and various instances of oxidative stress all influence the final end point. The existence of prodromal psychotic phases, structural-functional aspects of regional neuroimaging, dopamine signal overexpression, and psychosis propensity provide substance for neurodegenerative influences. The pathophysiology of schizophrenia spectrum disorders encompasses the destruction of normal functioning of the neurotrophins, in particular brain-derived neurotrophic factor (BDNF), dyskinesia of necessary ,ovements, and metabolic-metabolomic and proteomic markers. Neurotoxic accidents combined with genetic susceptibility appear to play a role in interfering with normal neurodevelopment or in tissue-destructive neurodegeneration or both, thereby elevating the eventual risk for disorder tendencies and eventual expression
650	7	a SAMHÄLLSVETENSKAPx Psykologi0 (SwePub)5012 hsv//swe
650	7	a SOCIAL SCIENCESx Psychology0 (SwePub)5012 hsv//eng
653		a GFK – Imaging – Infection – MAM – Perinatal – Reelin – Trophic factors – Vulnerability.
700	1	a Karilampi, Ullau Gothenburg University,Göteborgs universitet,Psykologiska institutionen,Department of Psychology4 aut0 (Swepub:gu)xkariu
700	1	a Ricci, S4 aut
700	1	a Rapp-Ricciardi, Max,d 1964u Gothenburg University,Göteborgs universitet,Psykologiska institutionen,Department of Psychology4 aut0 (Swepub:gu)xrapma
710	2	a Göteborgs universitetb Psykologiska institutionen4 org
773	0	t Handbook of Neurotoxicityd : Springerg , s. 2181-2205q <2181-2205z 9781461458357
856	4 8	u https://gup.ub.gu.se/publication/189037

Hitta via bibliotek

Handbook of Neurotoxicity (Sök värdpublikationen i LIBRIS)

Till lärosätets databas

Hitta mer i SwePub

Av författaren/redakt...: Archer, Trevor, ...; Karilampi, Ulla; Ricci, S; Rapp-Ricciardi, ...

Om ämnet

SAMHÄLLSVETENSKAP: SAMHÄLLSVETENSKA ...; och Psykologi

Artiklar i publikationen: Handbook of Neur ...

Av lärosätet: Göteborgs universitet

Sök utanför SwePub

Sök vidare i:: Google; Google Book Search; Google Scholar

Kungliga biblioteket hanterar dina personuppgifter i enlighet med EU:s dataskyddsförordning (2018), GDPR. Läs mer om hur det funkar här.
Så här hanterar KB dina uppgifter vid användning av denna tjänst.

LIBRIS.kb.se