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The Functional Polymorphism 844 A>G in Fc{alpha}RI (CD89) Does Not Contribute to Systemic Sclerosis or Rheumatoid Arthritis Susceptibility.
- Broen, Jasper C A (author)
- Coenen, Marieke J H (author)
- Rueda, Blanca (author)
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- Witte, Torsten (author)
- Padyukov, Leonid (author)
- Klareskog, Lars (author)
- Simeon, Carmen (author)
- Ortego-Centeno, Norberto (author)
- González-Gay, Miguel (author)
- Pros, Anna (author)
- Hunzelman, Nicholas (author)
- Riemekasten, Gabriela (author)
- Kreuter, Alexander (author)
- Vonk, Madelon (author)
- Scorza, Rafaella (author)
- Beretta, Lorenzo (author)
- Airò, Paulo (author)
- van Riel, Piet L C M (author)
- Kimberly, Robert (author)
- Martin, Javier (author)
- Edberg, Jeffrey (author)
- Radstake, Timothy R D J (author)
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- (creator_code:org_t)
- 2010-12-15
- 2011
- English.
- In: Journal of Rheumatology. - : The Journal of Rheumatology. - 0315-162X .- 1499-2752. ; 38:3, s. 446-449
- Journal article (peer-reviewed)
Abstract
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- OBJECTIVE: To investigate the role of the Fc(α)RI 844 A>G functional polymorphism in the genetic predisposition to rheumatoid arthritis (RA) and systemic sclerosis (SSc) susceptibility. METHODS: The study population was composed of 1401 patients with SSc, 642 patients with RA, and 1317 healthy controls. The Fc(α)RI (CD89) single-nucleotide polymorphism rs16986050 was genotyped by pyrosequencing. RESULTS: We observed no significant deviation of the genotype and allele frequencies in RA and SSc compared to controls. A metaanalysis and a recessive and dominant model yielded similar negative results. CONCLUSION: Our data show that the Fc(α)RI 844 A>G polymorphism is not associated with SSc or RA susceptibility.
Subject headings
- MEDICIN OCH HÄLSOVETENSKAP -- Klinisk medicin -- Reumatologi och inflammation (hsv//swe)
- MEDICAL AND HEALTH SCIENCES -- Clinical Medicine -- Rheumatology and Autoimmunity (hsv//eng)
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- art (subject category)
- ref (subject category)
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